A case of vertebrobasilar stroke during oxygen-ozone therapy

Cervical Spondylodiscitis after Oxygen–Ozone Therapy for Treatment of a Cervical Disc Herniation: A Case Report and Review of the Literature

“…Fort's group reported a spondylodiscitis in the lumbar spine after an intradiscal injection of oxygen-ozone caused by Achromobacter xylosoxidans [10]; Menendez P et al reported a Staphylococcus aureus paravertebral and intraabdominal abscess after six cycles of oxygen-ozone therapy [18]; and Bo et al reported the first and so far only case reported of a cervical spondylodiscitis with associated spinal epidural abscess (SEA) secondary to oxygen-ozone therapy for cervical disc herniation; some similarities with our case are noted including development of cervical infection with SEA and instability after debridement, and some differences like a sterility technique problem instead of a transesophageal puncture [4]. Other systemic complications reported are fulminant septicemia, a vertebrobasilar stroke, bilateral vitreo-retinal hemorrhages, and an unexpected death caused by gas embolism [7,11,15,17].…”

Anton syndrome during oxygen-ozone therapy

“…In that case, hypoperfusion of basilar artery was called Anton syndrome. Spontaneous bilateral infarct of posterior cerebral artery is commonly associated with embolism or a thrombus from basilar artery [4]. In our case, there was not any risk factor that could be associated with ischemic stroke, and our case is similar with the case that Corea et al [4] reported.…”

“…In 2004, Corea et al [4] reported a 66-year-old-woman who is the first case of ischemic stroke after medical oxygen-O 3 therapy. In that case, hypoperfusion of basilar artery was called Anton syndrome.…”

Anton–Babinski syndrome in an old patient: a case report and literature review

“…In addition to Anton–Babinski syndrome, other similar neuropsychiatric phenomena should also be considered when patients present with a similar condition . Similar neuropsychiatric phenomena include the following: akinetopsia, or motion blindness, in which patients can only see motionless objects but not moving ones; Riddoch syndrome, which manifests as residual motion vision within the hemianopic field; Charles Bonnet syndrome, which involves oriented, poor visual‐acuity patients having complex visual hallucinations; Balint's syndrome, which affects both visual perception, causing simultanagnosia and visual disorientation, and visual control of eye and hand movement, causing optic ataxia and ocular apraxia; prosopagnosia, a selective visual agnosia resulting from damage to a cortical network devoted to the accurate perception of faces; and Dide–Botcazo syndrome, an extension of Anton–Babinski syndrome but with involvement of memory circuitry, similar to the case report of temporary Anton–Babinski syndrome with amnesia . Our patient did not present with above conditions.…”