Acetylsalicylic acid (ASA)-augmentation in relation to ASA-intolerance

Paul, Eberhard; Gall, H.; Mechlin, A.; Möller, Ronald; Muller, I.

doi:10.1007/bf03360764

Cited by 4 publications

(7 citation statements)

References 8 publications

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“…5 In our three patients, many different therapeutic regimens were applied; however, no effective modality was achieved. In all cases, diagnosis of EV was based on histological examination, and polymerase chain reaction was performed from lesional skin specimens for the identification of HPV.…”

mentioning

confidence: 83%

“…5 In our three patients, many different therapeutic regimens were applied; however, no effective modality was achieved. The continuous administration of systemic low-dose isotretinoin may achieve prolonged remission status in EV.…”

mentioning

confidence: 83%

“…2 We recently observed a comparable patient who initially presented with NSAID hypersensitivity, developed chronic urticaria 4 years later and eventually lost both conditions after eradication of the intestinal protozoan Blastocystis hominis. 5 It can be speculated that here the allergic reaction is enhanced by pharmacologic actions of the NSAID (e.g. 4 In some food-allergic patients, systemic immediate type reactions develop only when they are exposed to the respective allergen and concomitantly to ASA.…”

Section: Loss Of Non-steroidal Anti-inflammatory Drug Hypersensitivitmentioning

confidence: 99%

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Loss of non‐steroidal anti‐inflammatory drug hypersensitivity after Helicobacter pylori eradication

Neynaber¹,

Berking²,

Przybilla³

2009

Acad Dermatol Venereol

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HAART). In all cases, diagnosis of EV was based on histological examination, and polymerase chain reaction was performed from lesional skin specimens for the identification of HPV.Patient 1 had the EV lesions for 96 months and was treated unsuccessfully with topical application of imiquimod for 2 months and cryosurgery for 6 months. Acitrecin and interferon a-2a were discontinued because of severe dryness and persistent flu-like symptoms, respectively. The administration of isotretinoin at a dose of 0.8 mg/kg proved efficacious for 6 months, leading to significant improvement; however, the lesions relapsed within 3 months after treatment cessation. HAART had no effect on EV lesions.Patient 2 developed EV and TBC and started HAART and a four-drug anti-TBC treatment (isoniazid, rifampicin, pyrazinamide and ethambutol). He did not consent in receiving any therapy for EV; nonetheless, a 50% improvement was noticed after initiation of HAART.Patient 3 under HAART presented widespread wart-like lesions distributed on the trunk, proximal extremities and resembling tinea versicolor. He was treated unsuccessfully with imiquimod and systemic isotretinoin, which was discontinued because of isotretinoin-induced headaches. He is currently under treatment with interferon a-2a with minimal prodromal results.There are either no large series or controlled studies of HIVassociated EV, and none are likely to be done probably because of the sparcity of cases. There is no established treatment for this dermatosis. 2 The role of HAART in EV remains questionable; 3 in our series, only one patient showed a significant improvement attributed to HAART.Acitrecin plus interferon alfa-2a, 2 etretinate 4 and isotretinoin 5 have shown transient effectiveness in the treatment of EV; recurrence has been noticed after the cessation of therapy. The continuous administration of systemic low-dose isotretinoin may achieve prolonged remission status in EV. 5 In our three patients, many different therapeutic regimens were applied; however, no effective modality was achieved. It seems that HIV-associated EV therapy probably has to be individualized, based primarily on the personal experience and preference of the investigators.

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mentioning

confidence: 83%

mentioning

confidence: 83%

Section: Loss Of Non-steroidal Anti-inflammatory Drug Hypersensitivitmentioning

confidence: 99%

See 1 more Smart Citation

Loss of non‐steroidal anti‐inflammatory drug hypersensitivity after Helicobacter pylori eradication

Neynaber¹,

Berking²,

Przybilla³

2009

Acad Dermatol Venereol

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“…Gut bekannt ist v. a. die keineswegs seltene nahrungsmittelallergieabhängige Anstrengungsanaphylaxie. Eine möglicherweise zu wenig beachtete Kombination von Auslösern ist die gleichzeitige Exposition gegenüber Allergenen und Azetylsalizylsäure, bei isoliertem Kontakt zu einer Einzelkomponente bleiben Symptome aus [17]. Als weitere, die Reaktionslage unter Umständen modifizierende Einflüsse sind beispielsweise Alkoholgenuss, körperliche oder psychische Stresssituationen oder bei Frauen die Zyklusphase zu berücksichtigen.…”

Section: äTiopathogeneseunclassified

Anaphylaxie

2007

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Anaphylaxis is an acute systemic hypersensitivity reaction with symptoms of immediate-type allergy, which involves particularly the skin, respiratory tract, cardiovascular system and gastrointestinal tract. The severity of anaphylactic reactions is variable but some are fatal. Hymenoptera venom anaphylaxis affects about 3% and food hypersensitivity 2.6-3.2% of the general population; drugs are the other frequent cause. Symptoms of anaphylaxis are characteristic, but none of them is obligatory--even urticaria is absent in about 10%--and each symptom may be found also in other conditions. Hence, there are numerous differential diagnostic considerations, and anaphylaxis may be overlooked in many cases. Diagnosis of anaphylaxis is based on the occurrence of characteristic symptoms, especially when they develop upon exposure to a potential trigger. It can be significantly supported by evidence of release of mediators in the course of the reaction. For clinical purposes, demonstration of an increase of mast cell tryptase serum concentration above the individual baseline value is useful. The correct diagnosis of anaphylaxis is not only important with regard to treatment of an acute reaction, but also for subsequent allergologic diagnostics and long-term management of the patient.

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“…Aze tyl sa li zyl säu re (ASS) ist nicht nur ein Ex azer ba ti ons-und Ag gra va ti ons fak tor ei ner aku ten oder chro ni schen Ur ti ka ria, son dern auch als Aug men ta ti ons fak tor einer IgE-ver mit tel ten Nah rungs mit tel aller gie be kannt [16]. Wei ter hin wur de beschrie ben, dass die Ein nah me von Aze tylsa li zyl säu re eine nah rungs mit tel ab hän gige an stren gungs in du zier te Ana phy la xie ver stär ken und zu ei nem po si ti ven Hauttest mit Nah rungs mit teln füh ren kann [1,9].…”

Section: Aze Tyl Sa LI Zyl Säu Reaug Men Ta Ti Onunclassified

Aktuelle Positionsbestimmung zur Bedeutung von Nahrungsmittelallergien und -unverträglichkeiten bei der Urtikaria

Wedi

Kapp

2006

Hautarzt

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Foods are usually the first suspect as the cause of urticaria. However, a causal relationship is found only in special subtypes of urticaria. IgE-mediated food allergy should be clearly separated from non-allergic hypersensitivity (pseudoallergic reactions). The former may play a role in acute urticaria, particularly in patients with atopic dermatitis. The responsible food proteins vary with age. IgE-mediated sensitization can apply to food-dependent exercise-induced urticaria/anaphylaxis but more often the combination of food intake (irrespective of which type) plus exercise results in symptoms. In chronic urticaria, IgE-mediated sensitization to food is normally irrelevant while pseudoallergic reactions to food additives and perhaps also to biogenic amines may be involved. Another urticaria subtype that may be caused by food is contact urticaria which is mostly found in the context of occupational food handling. Very rarely anisakiasis and nickel may cause food-induced urticaria. Aspirin is able not only to exacerbate and aggravate urticaria but can also enhance food-dependent urticaria.

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Acetylsalicylic acid (ASA)-augmentation in relation to ASA-intolerance

Cited by 4 publications

References 8 publications

Loss of non‐steroidal anti‐inflammatory drug hypersensitivity after Helicobacter pylori eradication

Loss of non‐steroidal anti‐inflammatory drug hypersensitivity after Helicobacter pylori eradication

Anaphylaxie

Aktuelle Positionsbestimmung zur Bedeutung von Nahrungsmittelallergien und -unverträglichkeiten bei der Urtikaria

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