Acute Inflammatory Conditions of the Uterine Adnexa

Viberg, Lars

doi:10.3109/00016346409156827

Cited by 28 publications

(3 citation statements)

References 32 publications

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“…The inflammatory process initiated by the gonococcus recruited initially aerobic and ultimately anaerobic bacteria (anaerobic progression) into the mechanistic aspects of disease [4,[8][9][10][11][12].…”

Section: Defining An Appropriate End Point For Therapymentioning

confidence: 99%

“…However, if the gonococcus was a constituent of a polymicrobial peritonitis, the therapeutic response was often altered, unless most of the organisms present were susceptible to the antibiotic regimen that was administered. Longterm follow-up showed that patients who had significantly altered therapeutic responses had a significantly higher incidence of secondary fertility than patients who had prompt responses [12][13][14]. When the anaerobic progression resulted in autoelimination of the gonococcus within the cul-de-sac, it was difficult to achieve a good therapeutic response with antibiotics whose primary spectrum of efficacy focused on N. gonorrhoeae and C. trachomatis.…”

Section: Defining An Appropriate End Point For Therapymentioning

confidence: 99%

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Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

Hemsel¹,

Ledger

Martens

et al. 2001

Clinical Infectious Diseases

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The International-Infectious Disease Society for Obstetrics and Gynecology-USA (I-IDSOG-USA) has concerns about the most recently published Centers for Disease Control and Prevention (CDC) guidelines for pelvic inflammatory disease (PID). I-IDSOG-USA advocates the following changes when the guidelines are revised. We recommend the use of the term "upper genital tract infection" (UGTI), followed by the designation of the etiologic agent, instead of the currently employed term, "pelvic inflammatory disease," or PID. In diagnoses, there should be greater emphasis on signs and symptoms related to subclinical or occult UGTI. Therapeutic recommendation for the treatment of UGTI should be documented for various stages of this diverse disease entity. There should be greater emphasis on hospitalization for infected nulligravida teenagers. This permits monitoring of antibiotic treatment and provides a site for medical educational efforts to teach this medically underserved segment of our society how to protect their future fertility, their health, and their lives.

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Section: Defining An Appropriate End Point For Therapymentioning

confidence: 99%

Section: Defining An Appropriate End Point For Therapymentioning

confidence: 99%

Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

Hemsel¹,

Ledger

Martens

et al. 2001

Clinical Infectious Diseases

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show abstract

“…hysteroophorectomized rat in vaginal oestrus phase induced by oestradiol. unilateral probability for two means to be identical (Hald 1960, Viberg 1964. parts per million.…”

Section: )mentioning

confidence: 99%

THE BEHAVIOUR OF INTRAVAGINALLY APPLIED Ag^110m‐LABELLED SILVER NITRATE

Nyberg

1967

Acta Obstet Gynecol Scand

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Treatment of Acute Pelvic Inflammatory Disease

Sweet¹

1997

Pelvic Inflammatory Disease

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Pelvic inflammatory disease (PID), one of the most common infections in nonpregnant women of reproductive age, remains an important public health problem. It is associated with major long-term sequelae, including tubal factor infertility, ectopic pregnancy, and chronic pelvic pain. In addition, treatment of acute PID and its complications incurs substantial health care costs. Prevention of these long-term sequelae is dependent upon development of treatment strategies based on knowledge of the microbiologic etiology of acute PID. It is well accepted that acute PID is a polymicrobic infection. The sexually transmitted organisms, Neisseria gonorrhoeae and Chlamydia trachomatis, are present in many cases, and microorganisms comprising the endogenous vaginal and cervical flora are frequently associated with PID. This includes anaerobic and facultative bacteria, similar to those associated with bacterial vaginosis. Genital tract mycoplasmas, most importantly Mycoplasma genitalium, have recently also been implicated as a cause of acute PID. As a consequence, treatment regimens for acute PID should provide broad spectrum coverage that is effective against these microorganisms.

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Acute Inflammatory Conditions of the Uterine Adnexa

Cited by 28 publications

References 32 publications

Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

THE BEHAVIOUR OF INTRAVAGINALLY APPLIED Ag^110m‐LABELLED SILVER NITRATE

Treatment of Acute Pelvic Inflammatory Disease

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Acute Inflammatory Conditions of the Uterine Adnexa

Cited by 28 publications

References 32 publications

Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

Concerns Regarding the Centers for Disease Control's Published Guidelines for Pelvic Inflammatory Disease

THE BEHAVIOUR OF INTRAVAGINALLY APPLIED Ag110m‐LABELLED SILVER NITRATE

Treatment of Acute Pelvic Inflammatory Disease

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THE BEHAVIOUR OF INTRAVAGINALLY APPLIED Ag^110m‐LABELLED SILVER NITRATE