Acute Quadriplegia From Hyperkalemia

Panichpisal, Kessarin; Gandhi, Shefali; Nugent, Kenneth; Anziska, Yaacov

doi:10.1097/nrl.0b013e3181b120b8

Cited by 21 publications

(15 citation statements)

References 47 publications

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“…[3] Consistent with these reports, our patient was receiving an angiotensin converting enzyme inhibitor (Ramipril), Beta-blocker (Atenolol) and a potassium sparing diuretic (Aldactone). In these case reports, potassium levels ranged from 8 mEq/L to 9.69 mEq/L[356] comparable to ours (9.1 mEq/L).…”

Section: Discussionsupporting

confidence: 76%

“…[123456] AFQ with no sensory deficit may mimic Guillain-Barré-syndrome (GBS). [7] Autonomic disturbance, a remarkable feature of GBS is not a recognized feature of hyperkalemic ascending quadriparesis, although it has been described in other situations associated with hyperkalemia.…”

Section: Discussionmentioning

confidence: 99%

“…The most frequently reported causes of hyperkalemia-induced AFQ are renal insufficiency,[2356] use of potassium-sparing drugs[12] or a combination of both. [3] One case was due to tumor lysis syndrome.…”

Section: Discussionmentioning

confidence: 99%

See 2 more Smart Citations

Hyperkalemia: A rare cause of acute flaccid quadriparesis

Garg

Saxena

Juneja

et al. 2014

Indian Journal of Critical Care Medicine

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Acute flaccid quadriparesis secondary to hyperkalemia is a very rare and serious but reversible medical emergency. We present a case of a 73-year-old female who was admitted with rapidly progressive ascending paraparesis progressing to quadriparesis in about 10 h due to hyperkalemia. Patient was treated with antihyperkalemic measures. Her power improved dramatically as potassium levels normalized and she had an uneventful recovery.

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Section: Discussionsupporting

confidence: 76%

Section: Discussionmentioning

confidence: 99%

See 1 more Smart Citation

Hyperkalemia: A rare cause of acute flaccid quadriparesis

Garg

Saxena

Juneja

et al. 2014

Indian Journal of Critical Care Medicine

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“…Cell membrane sodium channels can be inactivated through this prolonged and constant depolarization resulting in decreased membrane excitability that is exhibited by muscle weakness or paralysis. These symptoms can be manifested in cardiac and/or skeletal muscle [3, 5, 6]. …”

Section: Discussionmentioning

confidence: 99%

Acute Ascending Muscle Weakness Secondary to Medication-Induced Hyperkalemia

Kimmons

Usery

2014

Case Reports in Medicine

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Secondary hyperkalemic paralysis is an uncommon but potentially life-threatening consequence of drug-induced disease. We report a case of a 53-year-old female with history of chronic kidney disease presenting to the emergency department with a one-day history of upper and lower extremity weakness and paresthesias. Serum potassium concentration on admission was greater than 8 mEq/L, and serum creatinine was elevated above baseline. Electrocardiogram showed first-degree atrioventricular block with peaked T waves. The patient reported compliance with daily lisinopril 10 mg, spironolactone 25 mg, and 40 mEq twice daily of potassium chloride. Symptoms and electrocardiogram returned to baseline within 24 hours of presentation and serum potassium returned to 4.2 mEq/L at approximately 36 hours without the need for dialysis. This case emphasizes the importance of including such a condition in the differential diagnosis of patients with ascending paralysis and the importance of close monitoring of patients placed on potassium-elevating agents.

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“…[12] SHPP occurs in acute and chronic renal dysfunction, Addison's disease, rhabdomyolysis, excessive ingestion of potassium and drugs such as angiotensin converting enzyme inhibitors, potassium sparing diuretics, nonsteroidal anti-inflammatory drugs, cotrimoxazole, etc., Renal impairment is the most common cause, followed by Addison's disease and medications. [3]…”

Section: Discussionmentioning

confidence: 99%