Acute Torsion of a Wandering Spleen Causing Acute Abdomen

Yakan, Savaş; Telciler, KE; Denecli, AG

doi:10.1177/102490791101800107

“…Medical conditions complicated by splenic enlargement, including infectious mononucleosis and metabolic storage disorders predispose individual to splenic torsion in at risk groups (1,2). Conclusion Although rare, splenic torsion is an important differential diagnosis in patients presenting with acute abdomen of unclear aetiology.…”

mentioning

confidence: 97%

“…The patient underwent splenectomy. Discussion Splenic torsion is rare in children and is described in a condition referred to as wandering spleen (1) .Wandering spleen results from the abnormal development and absence of the normal splenic suspensory ligaments, resulting in an untethered organ predisposing to rotation and torsion of a large organ on a small vascular pedicle (1,2).…”

mentioning

confidence: 99%

See 1 more Smart Citation

P83 Evans syndrome with abdominal discomfort: a case report

Zia

¹

,

Ahmad

²

,

Ur

³

et al. 2019

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abdominal ultrasound was normal. C. difficile and C. Tropicalis were found in stool cultures. 2 consecutive sweat tests were performed, showing borderline values: 61,25 mmol/l; 67,7 mmol/l. There was no polyuria or excessive chloride loss in urine, which are typical features of Bartter's syndrome. Diarrhea was not present at birth, no watery stools were observed and the failure to thrive was not significant prior to admission, making the diagnosis of congenital chloride diarrhea unlikely. Genetic testing for CF showed that the infant was a delF508 mutation and G178r mutation compound heterozygote.The child showed no clinical symptoms of hypokalemia. No hyponatremia seizures were observed. Diarrhea was persistent. Rehydration therapy and antibiotic treatment significantly improved the condition.Patients who are compound heterozygous for the milder CFTR mutations (classes IV and V) experience partial CFTR function, resulting in atypical featuressuch as borderline sweat chloride concentrations, unlike our patient who is a compound heterozygous with class II and III mutation, where high amount of chloride in sweat is more commonly seen. Such patents are more likely to experience severe CF. Therefore making early diagnosis is crucial to further normal growth and development as therapy could be started early in life.

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“…Wandering spleen has two peaks of incidence the first in infancy with a male female ratio 2:1. Medical conditions complicated by splenic enlargement, including infectious mononucleosis and metabolic storage disorders predispose individual to splenic torsion in at risk groups (1,2). Conclusion Although rare, splenic torsion is an important differential diagnosis in patients presenting with acute abdomen of unclear aetiology.…”

mentioning

confidence: 97%

“…Prompt consideration should be given to this Diagnosis on account of the associated life threatening complication. Conservative management with preservation of the organ and splenoplexy is the treatment approach of choice but splenectomy is frequently necessary (2). Mortality risk relates chiefly to post-operative infection.…”

mentioning

confidence: 99%

P82 In pursuit of a wandering spleen

Canty

¹

,

Branagan

²

,

O’Halloran

³

et al. 2019

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0

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abdominal ultrasound was normal. C. difficile and C. Tropicalis were found in stool cultures. 2 consecutive sweat tests were performed, showing borderline values: 61,25 mmol/l; 67,7 mmol/l. There was no polyuria or excessive chloride loss in urine, which are typical features of Bartter's syndrome. Diarrhea was not present at birth, no watery stools were observed and the failure to thrive was not significant prior to admission, making the diagnosis of congenital chloride diarrhea unlikely. Genetic testing for CF showed that the infant was a delF508 mutation and G178r mutation compound heterozygote.The child showed no clinical symptoms of hypokalemia. No hyponatremia seizures were observed. Diarrhea was persistent. Rehydration therapy and antibiotic treatment significantly improved the condition.Patients who are compound heterozygous for the milder CFTR mutations (classes IV and V) experience partial CFTR function, resulting in atypical featuressuch as borderline sweat chloride concentrations, unlike our patient who is a compound heterozygous with class II and III mutation, where high amount of chloride in sweat is more commonly seen. Such patents are more likely to experience severe CF. Therefore making early diagnosis is crucial to further normal growth and development as therapy could be started early in life.

show abstract