2014
DOI: 10.1016/j.jsbmb.2013.11.009
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Adequate dietary vitamin D and calcium are both required to reduce bone turnover and increased bone mineral volume

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Cited by 21 publications
(12 citation statements)
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“…We also observed that 1% Ca supplement improved the BV/TV of the distal femur. It has been shown that salmon bone powder improved the BV/TV of OVX rats [29,30]. The observed increases in BV/TV, Tb.Th, Tb.N and Ct.Th, and the decrease in BSA/BV and Tb.Sp indicated an improved overall bone strength and tibial trabecular bone density.…”
Section: Discussionmentioning
confidence: 99%
“…We also observed that 1% Ca supplement improved the BV/TV of the distal femur. It has been shown that salmon bone powder improved the BV/TV of OVX rats [29,30]. The observed increases in BV/TV, Tb.Th, Tb.N and Ct.Th, and the decrease in BSA/BV and Tb.Sp indicated an improved overall bone strength and tibial trabecular bone density.…”
Section: Discussionmentioning
confidence: 99%
“…137 The genetic component is emerging as a strong predictor, as polymorphisms within various genes involved in the metabolic pathway of VitD (VitD-binding protein, megalin, cubilin, CYP27B1, CYP24A1, VDR) may explain not only a substantial percentage of the interindividual variability of 25-(OH)D, but also its association with major clinical outcomes 5,18,67,143,144 Several recently published reports challenge the established "normal" cut-off point for 25-(OH)D sufficiency. 19,21 According to the NHANES 2005-2006 findings, the mean 25-(OH)D concentration is approximately 24 ng/mL in most age groups. Furthermore, the US Institute of Medicine (IOM) in 2011 concluded that serum 25-(OH)D levels >20 ng/mL are sufficient for almost the entire (97.5%) population, at least as regards Caucasians.…”
Section: 55mentioning
confidence: 99%
“…11 This rationale is based on maximal intestinal calcium absorption and nadir concentrations of parathyroid hormone (PTH), reported to be achievable at 25-(OH)D levels of approximately 30-40 ng/mL, [12][13][14][15] and is underscored by the updated recommendations of the US Endocrine Society. 1 However, both tenets have recently been challenged: [16][17][18][19] The boundary for clinically relevant VitD deficiency, characterized by osteopenia and rickets/ osteomalacia, has traditionally been set at 10 ng/mL 25-(OH)D, 17,20 a point recently disputed by the US Endocrine Society, that has raised the VitD deficiency threshold to 20 ng/mL 25-(OH)D. 1 25-(OH)D "insufficiency" spans the range between the frank deficiency threshold and the lower normal level (traditionally 10-30 ng/mL), indicating inadequate stores for optimal VitD functions, but generally not associated with overt clinical symptoms. 21 VitD overall status in vivo is best estimated by the concentration of serum total 25-(OH)D, i.e.…”
Section: Introductionmentioning
confidence: 99%
“…Furthermore, what is also essential is the para-and autocrine control by cytokines, growth factors and via the nervous system (Togari et al 2008) The literature has well documented the relationships between bone mineral density (BMD) and bone mass (BMC), and vitamin D and calcium (Uusi-Rasi et al 2013;Lee et al 2014). Some studies point to another important relationship between the state of bone tissue (the occurrence of osteoporosis), and the lipid level in the blood serum (Brownbill et al 2006;Ghadiri-Anari et al 2016).…”
Section: Introductionmentioning
confidence: 99%