An Australian Exclusion Diet

Gibson, Anne R.; Clancy, R. L.

doi:10.5694/j.1326-5377.1978.tb112553.x

Cited by 10 publications

(4 citation statements)

References 4 publications

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“…In 1978, the Australian pioneers in this field published a summary article on the application of an elimination diet for the management of chronic urticaria [Gibson and Clancy, 1978]. The basis for the elimination diet is still the same today: following a strict exclusion diet for 2–4 weeks, followed by dietary challenges to determine which of the components are contributing to symptoms.…”

Section: The Elimination Dietmentioning

confidence: 99%

Fermentable oligosaccharides, disaccharides, monosaccharides and polyols (FODMAPs) and nonallergic food intolerance: FODMAPs or food chemicals?

Barrett

Gibson

2012

Therap Adv Gastroenterol

131

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Food intolerance in irritable bowel syndrome (IBS) is increasingly being recognized, with patients convinced that diet plays a role in symptom induction. Evidence is building to implicate fermentable oligosaccharides, disaccharides, monosaccharides and polyols (FODMAPs) in the onset of abdominal pain, bloating, wind and altered bowel habit through their fermentation and osmotic effects. Hypersensitivity to normal levels of luminal distension is known to occur in patients with IBS, with consideration of food chemical intolerance likely to answer many questions about this physiological process. This paper summarizes the evidence and application of the most common approaches to managing food intolerance in IBS: the low-FODMAP diet, the elimination diet for food chemical sensitivity and others including possible noncoeliac gluten intolerance.

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Section: The Elimination Dietmentioning

confidence: 99%

Fermentable oligosaccharides, disaccharides, monosaccharides and polyols (FODMAPs) and nonallergic food intolerance: FODMAPs or food chemicals?

Barrett

Gibson

2012

Therap Adv Gastroenterol

131

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“…The RPAH diet was first described in 1978 to treat chronic urticaria [ 68 ] and subsequently to manage gastrointestinal symptoms due to food intolerances [ 65 ]. There are no published observational or interventional studies of the RPAH to treat IBS, although there is commentary in the literature [ 61 , 69 , 70 ].…”

Section: Dietary Management Of Chronic Diarrhoeamentioning

confidence: 99%

What Are the Pearls and Pitfalls of the Dietary Management for Chronic Diarrhoea?

et al. 2021

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Chronic diarrhoea affects up to 14% of adults, it impacts on quality of life and its cause can be variable. Patients with chronic diarrhoea are presented with a plethora of dietary recommendations, often sought from the internet or provided by those who are untrained or inexperienced. In this review, we summarise the possible causes of chronic diarrhoea that can be managed by diet, the symptom improvement and quality of life benefits but also the potential risks of such dietary treatments. Clinicians need to consider both the benefits and risks of dietary treatments before making dietary recommendations to manage chronic diarrhoea. The pivotal role that dietitians have in ensuring optimal symptom improvement without jeopardising nutritional and overall health is discussed.

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“…The study was approved by the institution's Ethics and Pharmacy Committees. Patients with chronic urticaria of more than 3 months duration were subdivided into ASA and additive sensitive and non-sensitive groups on the basis of history (urticaria induced by ASA or tartrazine containing foods), challenge with ASA or tartrazine, and response to an additive-free diet [15]. All such patients were receiving regular antihistamines and a proportion (four out of twelve) took oral steroids intermittently for severe exacerbations.…”

Section: Subjectsmentioning

confidence: 99%

Cell‐mediated immune responses to artificial food additives in chronic urticaria

Warrington

Sauder

McPhillips

1986

Clin Experimental Allergy

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Abstract. In some cases of chronic urticaria it is suspected that food additives such as tartrazine and sodium benzoate or salicylates may play a role in the pathogenesis of the condition. Since, at times, chronic urticaria may appear histologically similar to a mild cell‐mediated immune response, the release of the T cell‐derived lymphokine leucocyte inhibitory factor (LIF), in response to incubation with these additives and with acetylsalicylic acid (ASA), was measured in vitro using cells from normal controls, from patients with chronic urticaria with or without clinically associated additive sensitivity and from patients with asthma with or without associated ASA sensitivity. It was found that significant production of LIF occurred in response to tartrazine and sodium benzoate in those individuals with chronic additive induced urticaria. In addition, tartrazine caused LIF release from mononuclear cells of ASA‐sensitive asthmatics. These results may indicate a possible role for additive‐induced cell‐mediated immune responses in the pathogenesis of some cases of chronic urticaria and suggest a potential diagnostic test for this condition.

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An Australian Exclusion Diet

Cited by 10 publications

References 4 publications

Fermentable oligosaccharides, disaccharides, monosaccharides and polyols (FODMAPs) and nonallergic food intolerance: FODMAPs or food chemicals?

Fermentable oligosaccharides, disaccharides, monosaccharides and polyols (FODMAPs) and nonallergic food intolerance: FODMAPs or food chemicals?

What Are the Pearls and Pitfalls of the Dietary Management for Chronic Diarrhoea?

Cell‐mediated immune responses to artificial food additives in chronic urticaria

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