1986
DOI: 10.1016/0022-1759(86)90272-3
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An enzyme-linked immunosorbent assay for differential quantitation of secretory immunoglobulins of the A and M isotypes in human serum

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Cited by 44 publications
(19 citation statements)
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“…65]. The very high prevalence of abnormal values of S-IgA was already noted in the first assess ments of S-IgA in LD: it was higher than that observed in chronic LD (80 vs. 50%), even in ALD [58], More sophisticated studies have not only confirmed these results but have also shown that very high values (up to 200 pg/ml SC) could be observed [46]; they also showed that SC could be bound both to plgA and IgM; binding of IgM was particu larly important in type A-ALI [59,61], re flecting the total IgM increase which is usually present in this disease. These obser vations have brought new arguments in fa vor of a liver origin of serum SC; moreover the follow-up of cases of fulminant hepatitis with spontaneous recovery gave additional evidence for the cellular localization of SC in the regenerating liver [65], In AH, S-IgA cor related strongly with serum aspartate amino transferase and, to a lesser extent, with leu cine aminopeptidase and alkaline phospha tase activities but not with direct bilirubin levels.…”
Section: Iga In Acute Liver Necrosis (Acute and Fulminant Hepatitis)mentioning
confidence: 49%
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“…65]. The very high prevalence of abnormal values of S-IgA was already noted in the first assess ments of S-IgA in LD: it was higher than that observed in chronic LD (80 vs. 50%), even in ALD [58], More sophisticated studies have not only confirmed these results but have also shown that very high values (up to 200 pg/ml SC) could be observed [46]; they also showed that SC could be bound both to plgA and IgM; binding of IgM was particu larly important in type A-ALI [59,61], re flecting the total IgM increase which is usually present in this disease. These obser vations have brought new arguments in fa vor of a liver origin of serum SC; moreover the follow-up of cases of fulminant hepatitis with spontaneous recovery gave additional evidence for the cellular localization of SC in the regenerating liver [65], In AH, S-IgA cor related strongly with serum aspartate amino transferase and, to a lesser extent, with leu cine aminopeptidase and alkaline phospha tase activities but not with direct bilirubin levels.…”
Section: Iga In Acute Liver Necrosis (Acute and Fulminant Hepatitis)mentioning
confidence: 49%
“…Such observations were confirmed using more quantitative techniques, such as radioimmunoassays [46. 57], or ELISA [59,60], However, no correlation was found be tween plgA. IgA2, or total IgA and total SC concentrations (considered in most studies as reflecting S-IgA), in any particular group of patients with LD, including ALD [46], In ALD, the mean S-IgA values were always lower than those observed in biliary tract obstruction, acute hepatitis (AH) or primary hepatic tumors, and they were of the same range as those measured in chronic active LD of nonalcoholic origin.…”
Section: Iga and Chronic Alcoholic Liver Diseasementioning
confidence: 99%
“…In serum, SC is always complexed with pIgA and pIgM (Brandtzaeg, 1985); release of SC from positive tumours into blood may therefore raise the serum levels of secretory IgA (SIgA) and secretory IgM (SIgM) which are normally circulating only in trace amounts (Kvale & Brandtzaeg, 1986). Nevertheless, we recently found generally low serum concentrations of total SC in patients with colonic (Kvale et al, 1987a) or breast carcinomas (Kvale et al, 1987c).…”
mentioning
confidence: 98%
“…Serum values were determined in relation to appropriate SIgA or SIgM standards. The total concentration of circulating SC was calculated from the SIgA and SIgM values obtained in each individual (Kvale & Brandtzaeg, 1986). Sera from 71 sex-and age-matched healthy subjects were included as controls (age, 29-91 years).…”
mentioning
confidence: 99%
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