1969
DOI: 10.1016/s0016-5085(19)33962-9
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An Experience of Ulcerative Colitis

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Cited by 193 publications
(19 citation statements)
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“…Due to misdiagnosis patient received non-specific treatment. However, clinical signs and symptoms in our case and findings of laboratory investigations were consistent with diagnostic criterion of toxic megacolon [14].…”
Section: Discussionsupporting
confidence: 87%
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“…Due to misdiagnosis patient received non-specific treatment. However, clinical signs and symptoms in our case and findings of laboratory investigations were consistent with diagnostic criterion of toxic megacolon [14].…”
Section: Discussionsupporting
confidence: 87%
“…In our case, the outcome of pregnancy was uneventful; however, severe colitis developed in puerperal period and a risk factor for such factor remains undetermined. The hallmark of clinical diagnostic criteria of toxic megacolon as suggested by Jalan et al [14] are (i) radiographic evidence of colonic dilatation (more than 6 cm in transverse colon), (ii) any 3 of these (fever > 101.5°F, tachycardia > 120 beats/min, leucocytosis> 10.5 x 10 3 /µL, anemia), (iii) any one of these (dehydration, altered mental status, electrolyte abnormality, hypotension). Similarly, in our case; we noticed presence of all the clinical diagnostic criterion of toxic megacolon except non-specific USG finding as described above.…”
Section: Discussionmentioning
confidence: 99%
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“…Of this number, 11 patients were definitely not in the first attack of their disease; this finding is similar to that of Jalan, Sircus, Card, Falconer, Bruce, Crean, McManus, Small, and Smith (1969a) who found that, of 16 patients who sustained perforation, 11 did so during a relapse and only five in their first attack. Other authors (Edwards and Truelove, 1964;de Dombal, Watts, Watkinson, and Goligher, 1965) have found perforation to be more common during the initial attack of ulcerative colitis.…”
Section: Discussionsupporting
confidence: 84%
“…In critically ill patients with severe conflicts the inflammation may even pass through the mucous layer and reach the muscle layer. In this condition intestinal motility has been impaired, bowel movements decreased and the patient may go into the toxic mega colon [ 4 , 5 ]. The disease was introduced first by Wills in London as a separate disease from clinical diarrhea [ 6 ].…”
Section: Introductionmentioning
confidence: 99%