An experimental test of the effect of acute anxious arousal and anxiety sensitivity on negative reinforcement smoking

Farris, Samantha G.; Zvolensky, Michael J.

doi:10.1177/0269881116642880

Cited by 16 publications

(25 citation statements)

References 58 publications

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“…Non-treatment seeking adult daily smokers were recruited for an experimental study on “smoking behavior” (Farris and Zvolensky, 2016). Community-recruited individuals who were between 18–65 years of age, reported smoking 10 or more cigarettes per day for at least one year, and smoked within the first 30 minutes of waking in the morning, were invited for a baseline assessment to determine eligibility for the experimental study (Farris and Zvolensky, 2016).…”

Section: Methodsmentioning

confidence: 99%

“…Community-recruited individuals who were between 18–65 years of age, reported smoking 10 or more cigarettes per day for at least one year, and smoked within the first 30 minutes of waking in the morning, were invited for a baseline assessment to determine eligibility for the experimental study (Farris and Zvolensky, 2016). Participants were excluded from participation during an initial telephone screen if they reported frequent drinking (≥ 9 standard drinks/week), illicit drug use (≥ 3 days/week), unstable medical conditions, or current psychotic symptoms.…”

Section: Methodsmentioning

confidence: 99%

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Tobacco demand, delay discounting, and smoking topography among smokers with and without psychopathology

Farris

Aston

Abrantes

et al. 2017

Drug and Alcohol Dependence

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Introduction Tobacco demand (i.e., relative value attributed to a given reinforcer) and delay discounting (i.e., relative preference for smaller immediate rewards over larger delayed rewards) are two behavioral economic processes that are linked to the progression of problematic substance use. These processes have not been studied among those with psychopathology, a vulnerable group of smokers. The current study examined differences in tobacco demand and delay discounting, and their association with smoking topography among smokers with (n=43) and without (n=64) past-year psychopathology. Method Adult daily smokers (n = 107, Mage = 43.5; SD = 9.7) participated in a study on “smoking behavior.” Past-year psychological disorders were assessed via a clinician-administered diagnostic assessment. All subjects participated in an ad libitum smoking trial and then completed an assessment of delay discounting (Monetary Choice Questionnaire) and tobacco demand (Cigarette Purchase Task) approximately 45–60 minutes post-smoking. Results Smokers with psychopathology, compared to those without, had significantly higher demand intensity and maximum expenditure on tobacco (Omax), but did not differ on other demand indices or delay discounting. Smokers with psychopathology had shorter average inter-puff intervals and shorter time to cigarette completion than smokers without psychopathology. Tobacco demand and delay discounting measures were significantly intercorrelated among smokers with psychopathology, but not those without. Both behavioral economic measures were associated with specific aspects of smoking topography in smokers with psychopathology. Discussion The association between tobacco demand and delay discounting is evident among smokers with psychopathology and both measures were most consistently related to smoking behavior.

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Section: Methodsmentioning

confidence: 99%

Section: Methodsmentioning

confidence: 99%

Tobacco demand, delay discounting, and smoking topography among smokers with and without psychopathology

Farris

Aston

Abrantes

et al. 2017

Drug and Alcohol Dependence

View full text Add to dashboard Cite

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“…Exposure to adverse life experiences may contribute to increased negative affect (Kim et al 2013; Young et al 2007), which may, in turn, increase vulnerability to and risk for maintenance of addictive behaviors (Farris and Zvolensky 2016; Muscatello et al 2016). Large epidemiological, cross-sectional, and, more recently, prospective studies have documented a link between early exposure to adversity and smoking (Bellis et al 2014a; Edwards et al 2007; Elliott et al 2014; Ford et al 2011; Fuller-Thomson et al 2013; Xie et al 2012).…”

Section: Introductionmentioning

confidence: 99%

Early life adversity influences stress response association with smoking relapse

et al. 2017

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Rationale We examined the hypothesis that stress-related blunting of cortisol in smokers is particularly pronounced in those with history of severe life adversity. Objectives The 2 aims of this study were first to examine hormonal, craving, and withdrawal symptoms during ad libitum smoking and after the first 24 hours of abstinence in smokers who experienced high or low levels of adversity. Secondly, we sought to examine the relationship between adversity and HPA hormones to predict relapse during the first month of a smoking cessation attempt. Methods Hormonal and self-report measures were collected from 103 smokers (49 women) during ad libitum smoking and after the first 24 hours of abstinence. Hypothalamic-pituitary-adrenal (HPA) hormones were measured during baseline rest and in response to acute stress in both conditions. All smokers were interested in smoking cessation, and we prospectively used stress response measures to predict relapse during the first four weeks of the smoking cessation attempt. Results The results showed that high adversity was associated with higher distress and smoking withdrawal symptoms. High level of early life adversity was associated with elevated HPA activity, which was found in both salivary and plasma cortisol. Enhanced adrenocorticotropic hormone (ACTH) stress response was evident in high adversity but not in low adversity relapsers. Conclusions This study demonstrated that early life adversity is associated with stress-related HPA responses. The study also demonstrated that, among smokers who experienced a high level of life adversity, heightened ACTH and cortisol responses were linked with increased risk for smoking relapse.

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“…Adult daily smokers were recruited for an experimental study on “smoking behavior” (Farris & Zvolensky, 2016) via flyers posted in community areas and college campuses, newspaper advertisements, webpage announcements (e.g. university website, Craigslist.com), and word-of-mouth.…”

Section: Methodsmentioning

confidence: 99%

“…university website, Craigslist.com), and word-of-mouth. Community-recruited smokers who were between 18 and 65 years of age, reported smoking 10 or more cigarettes per day for at least one year, and smoked within the first 30 min of waking in the morning, were invited for a baseline assessment to determine eligibility for the experimental study (described in Farris & Zvolensky, 2016). The current study is a secondary analysis of data from all participants who completed the baseline assessment, regardless of if they were deemed eligible for the experimental phase of the study.…”

Section: Methodsmentioning

confidence: 99%

Emotion dysregulation explains the relation between insomnia symptoms and negative reinforcement smoking cognitions among daily smokers

Kauffman

Farris

Alfano

et al. 2017

Addictive Behaviors

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Introduction Insomnia co-occurs with smoking. However, mechanisms that may explain their comorbidity are not well known. Method The present study tested the hypothesis that insomnia would exert an indirect effect on negative reinforcement smoking processes via emotion dysregulation among 126 adult non-treatment seeking daily smokers (55 females; Mage = 44.1 years, SD = 9.72). Negative reinforcement smoking processes included negative reinforcement smoking outcome expectancies, negative reinforcement smoking motives, and two negative expectancies from brief smoking abstinence (somatic symptoms and harmful consequences). Results Insomnia symptoms yielded a significant indirect effect through emotion dysregulation for negative reinforcement smoking outcome expectancies, negative reinforcement smoking motives, and harmful consequences expectancies from brief smoking abstinence. In contrast to prediction, however, insomnia was not associated with somatic symptom expectancies from brief smoking abstinence through emotion dysregulation. Conclusions These data may suggest that the indirect effect of emotion dysregulation is more relevant to cognitive-affective negative reinforcement processes rather than somatic states. Overall, the present findings contribute to a growing body of literature linking emotion dysregulation as an explanatory mechanism for insomnia and smoking and uniquely extend such work to an array of clinically significant negative reinforcement smoking processes.

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An experimental test of the effect of acute anxious arousal and anxiety sensitivity on negative reinforcement smoking

Cited by 16 publications

References 58 publications

Tobacco demand, delay discounting, and smoking topography among smokers with and without psychopathology

Tobacco demand, delay discounting, and smoking topography among smokers with and without psychopathology

Early life adversity influences stress response association with smoking relapse

Emotion dysregulation explains the relation between insomnia symptoms and negative reinforcement smoking cognitions among daily smokers

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