1997
DOI: 10.1097/00005373-199703000-00015
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Analysis of 372 Patients with Crush Syndrome Caused by the Hanshin-Awaji Earthquake

Abstract: Peak serum concentration of creatine kinase as well as the number of injured extremities serve to estimate the severity of crush syndrome.

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Cited by 218 publications
(167 citation statements)
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“…The incidence probably higher than that we observed. In other studies, the incidence of Crush syndrome rages from 3.9 to 5.4 % Oda et al 1997).…”
Section: The Work Of the Team In Yaanmentioning
confidence: 77%
“…The incidence probably higher than that we observed. In other studies, the incidence of Crush syndrome rages from 3.9 to 5.4 % Oda et al 1997).…”
Section: The Work Of the Team In Yaanmentioning
confidence: 77%
“…[14,15] • Crush syndrome was defined in patients as crush injury with systemic manifestations. [10] • Patients with crush injury and either oliguria (urinary output <400 mL/d), elevated levels of BUN (>40 mg/ dL), or serum creatinine (>2.0 mg/dL) were considered as patients with renal impairment.…”
Section: Methodsmentioning
confidence: 99%
“…[9] Moreover the pressure causes necrosis of the muscle, and during revascularization, the diffusion of calcium, sodium, and water into the damaged muscle cellstogether with the loss of potassium, phosphate, lactic acid, myoglobin, and creatinine kinase-can lead to hyperkaliemia and acidosis. [10][11][12][13][14] Thus, the renal problems due to pressure-induced rhabdomyolysis have been claimed to carry unique laboratory features as compared to other causes of ARF, such as increased serum levels of muscle enzymes and a higher ratio of creatinine/blood urea nitrogen (BUN), as well as higher degrees of uricemia, phosphatemia and, most importantly, life-threatening hyperkaliemia. [8] When these laboratory abnormalities come together with a high incidence of surgical and medical complications, [13] calculated mortality rates of up to 40% in dialyzed patients can be expected.…”
Section: Introductionmentioning
confidence: 99%
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