1972
DOI: 10.1161/01.cir.46.1.14
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Analysis of Left Ventricular Wall Motion by Reflected Ultrasound

Abstract: Ultrasound echocardiograms from the septal and posterior left ventricular walls were displayed with a simultaneously recorded electrocardiogram, phonocardiogram, and indirect carotid pulse. These echoes differed in both amplitude and waveform. The contour of the posterior wall echo resembled an inverted ventricular volume curve, while the septal echo was of smaller amplitude and had a characteristic notched appearance. Most of the movement of the left ventricular walls relative to the ultrasound transducer was… Show more

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Cited by 208 publications
(48 citation statements)
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“…1'1 Second, even if septal curvature correction continued throughout systole, its tendency to produce net anterior septal motion would be progressively attenuated by the systolic reduction in left ventricular dimensions, an effect that is maximal in midsystole. 18 This hypothesis implies that the septum moves toward the center of the left ventricle after early systole in patients with RVVO. 16 If this is so, the M mode echocardiographic appearance of anterior motion, or no net motion, of the septum during mid-late systole2' 7 could be explained only by anterior motion of the entire left ventricle during this period.…”
mentioning
confidence: 99%
“…1'1 Second, even if septal curvature correction continued throughout systole, its tendency to produce net anterior septal motion would be progressively attenuated by the systolic reduction in left ventricular dimensions, an effect that is maximal in midsystole. 18 This hypothesis implies that the septum moves toward the center of the left ventricle after early systole in patients with RVVO. 16 If this is so, the M mode echocardiographic appearance of anterior motion, or no net motion, of the septum during mid-late systole2' 7 could be explained only by anterior motion of the entire left ventricle during this period.…”
mentioning
confidence: 99%
“…For instance, systolic wall motions at the center of infarctions were mostly paradoxical and systolic thickening was never seen. The latter finding is fundamental in differentiating ischemia and infarction from hypokinetic or even paradoxical regional wall motions caused by more physiological phenomena such as septal asynergy in the right ventricular volume overload (Corya et al, 1977), and in physically highly trained subjects (Nieminen, 1977a), from bundle-branch block, or simply due to the anterior mass motion of the whole heart seen normally in systole (McDonald et al, 1972). Loss of systolic thickening together with reduced or absent systolic motion amplitude is one of the characteristic forms of asynergy in experimental myocardial ischemia or infarction (Heikkila et al, 1972;Kerber and Marcus, 1978;Ross and Franklin, 1976;Sasayama et al, 1976).…”
Section: Discussionmentioning
confidence: 99%
“…Transducer position, echographic gain, and patient position were adjusted to obtain optimal recordings of left ventricular dimensions in the minor axis. 12 In all cases repeat echocardiograms were obtained with the patient in the same position as for the baseline study.…”
Section: Methodsmentioning
confidence: 99%