Anton's Syndrome in a Patient With Posttraumatic Optic Neuropathy and Bifrontal Contusions

McDaniel, Keith D.; McDaniel, Leslie D.

doi:10.1001/archneur.1991.00530130113028

Cited by 31 publications

(14 citation statements)

References 23 publications

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“…Anton–Babinski syndrome is one of specific form of cortical blindness. The aetiologies include the following: ischemia stroke or haemorrhage involving occipital lobes; posterior leukoencephalopathy induced by chemotherapy, radiotherapy, preeclampsia, adrenoleukodystrophy, or mitochondrial encephalomyopathy, lactic acidosis, and stroke‐like episodes; obstetric haemorrhage with posterior circulation hypoperfusion; and trauma‐related optic neuropathy, bifrontal contusions, and callosal disconnection . Recovery of visual function can be expected if the underlying factor is posterior reversible leukoencephalopathy or cortical hypoperfusionis, and it is corrected quickly .…”

Section: Discussionmentioning

confidence: 99%

Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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Anton-Babinski syndrome is a rare disease featuring bilateral cortical blindness and anosognosia with visual confabulation, but without dementia or any memory impairment. It has a unique neuropsychiatric presentation and should be highly suspected in those with odd visual loss and imaging evidence of occipital lobe injury. In the case discussed herein, a 90-year-old man presented with bilateral blindness, obvious anosognosia, and vivid visual confabulation, which he had had for 3 days. Brain computed tomography demonstrated recent hypodense infarctions at the bilateral occipital lobes. Thus, the patient was diagnosed with Anton-Babinski syndrome. Because of his age and the thrombolytic therapy during the golden 3 hours after ischemic stroke, the patient received aspirin therapy rather than tissue plasminogen activator or warfarin. He gradually realized he was blind during the following week, but died of pneumonia 1 month later. In the literature, it is difficult to establish awareness of blindness in patients with Anton-Babinski syndrome, but optimistically, in one report, a patient was aware of blindness within 2 weeks, without vision improvement. Our case illustrates that elderly patients with Anton-Babinski syndrome can partially recover and that 1 week is the shortest time for the establishment of awareness of blindness for sufferers without vision improvement.

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Section: Discussionmentioning

confidence: 99%

Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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“…The literature provides multiple indirect evidence of a neural basis for a number of phenomenological variables of AVH, including: AVH-anosognosia (unawareness of the perception-object dissociation), content (systematized or repetitive), space location (inner or outer space), and familiarity and the gender of the “voices.” Anosognosia of neurological symptoms (e.g., cortical blindness, and left side hemiparesis) is associated with symptom-specific neural correlates—lesions of the visual associative cortex (Magitot and Hartmann, 1926) or frontal lobes (McDaniel and McDaniel, 1991) in the case of cortical blindness, and lesions in the non-dominant motor cortex in the case of left side hemiplegia (Babinski, 1914). As AVH in the clinical population are symptoms of brain disease just like blindness or hemiplegia, AVH-anosognosia could be associated with specific neural substrates (Stephane et al, 2003).…”

Section: The Neural Basis Of Avh Phenomenologymentioning

confidence: 99%

Auditory verbal hallucinations result from combinatoric associations of multiple neural events

Stephane¹

2013

Front. Hum. Neurosci.

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While Auditory Verbal Hallucinations (AVH) refer to specific experiences shared by all subjects who have AVH—the perception of auditory speech without corresponding external stimuli, the characteristics of these experiences differ from one subject to another. These characteristics include aspects such as the location of AVH (inside or outside the head), the linguistic complexity of AVH (hearing words, sentences, or conversations), the range of content of AVH (repetitive or systematized content), and many other variables. In another word, AVH are phenomenologically heterogeneous experiences. After decades of research focused on a few explanatory mechanisms for AVH, it is apparent that none of these mechanisms alone explains the wide phenomenological range of AVH experiences. To date, our phenomenological understanding of AVH remains largely disjointed from our understanding of the mechanisms of AVH. For a cohesive understanding of AVH, I review the phenomenology and the cognitive and neural basis of AVH. This review indicates that the phenomenology of AVH is not a pointless curiosity. How a subject describes his AVH experiences could inform about the neural events that resulted in AVH. I suggest that a subject-specific combinatoric associations of different neural events result in AVH experiences phenomenologically diverse across subjects.

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“…Other diseases described as causes are MELAS [8], preeclampsia [2], obstetric hemorrhage [9], trauma [10], adrenloeucodistrophy [11], hypertensive encephalopathy [12] and angiographic procedures [13].…”

Section: Review Of the Literaturementioning

confidence: 99%

Visual Anosognosia (Anton-Babinski Syndrome): Report of Two Cases Associated with Ischemic Cerebrovascular Disease

Carvajal¹,

Cárdenas²,

Pazmiño³

et al. 2012

JBBS

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Visual anosognosia or Anton-Babinski syndrome is a rare neurological condition related to cortical blindness. The patients deny their blindness and affirm adamantly that they are capable of seeing. The clinical presentation includes confabulations and sometimes confusional states. In this article we report two patients with anosognosia related to ischemic stroke in two different sets of etiology and pathogenesis. We describe the major clinical manifestations of this syndrome and review the current medical literature. Two patients were identified, a 96-year-old male with visual anosognosia secondary to a right posterior cerebral artery thrombosis, and a 56-year-old female with the same syndrome but related to central nervous system angiitis in relation with multiple sclerosis and Hashimoto's thyroiditis. Visual anosognosia or Anton-Babinski syndrome is a rare neurological condition. However the ischemic vascular cerebral disease is a frequent etiology. We believe that this is the first report of this syndrome in relation to angiitis with a clear autoimmune pathogenesis.

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Anton's Syndrome in a Patient With Posttraumatic Optic Neuropathy and Bifrontal Contusions

Cited by 31 publications

References 23 publications

Anton–Babinski syndrome in an old patient: a case report and literature review

Anton–Babinski syndrome in an old patient: a case report and literature review

Auditory verbal hallucinations result from combinatoric associations of multiple neural events

Visual Anosognosia (Anton-Babinski Syndrome): Report of Two Cases Associated with Ischemic Cerebrovascular Disease

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