Apical thrombus in tako-tsubo cardiomyopathy

Schmidt, Michael; Herholz, C; Block, Malcolm

doi:10.1136/bcr.2006.100941

Cited by 4 publications

(3 citation statements)

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“…Cardiac MRI was usefully performed in 4 studies [12,16,17,34], whereas ventriculography alone in another one [27]. A 4 hour time interval was the cut-off considered as the maximum time needed to perform TTE soon after hospitalization (n = 9 patients).…”

Section: Thrombus Morphologymentioning

confidence: 99%

Cardioembolic outcomes in stress-related cardiomyopathy complicated by ventricular thrombus: A systematic review of 26 clinical studies

Gregorio

2010

International Journal of Cardiology

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Section: Thrombus Morphologymentioning

confidence: 99%

Cardioembolic outcomes in stress-related cardiomyopathy complicated by ventricular thrombus: A systematic review of 26 clinical studies

Gregorio

2010

International Journal of Cardiology

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“…Basierend auf der kontraktilen Dysfunktion mit Folgen der Akinesie besteht eine erhöhte Thrombogenität und damit ein gesteigertes Risiko bezüglich der (Neu)Entstehung von intraventrikulären Thromben mit Embolisationsgefahr [9]. Neben der mechanischen Prädisposition (verstärkte Wandhaftung der Thrombozyten durch die fast stehende apikale Wandregion) werden thrombozytäre Dysfunktionen mitverantwortlich gemacht.…”

Section: Therapie Und Verlaufunclassified

“…Im Rahmen des Tako-TsuboSyndroms mit komplizierendem linksventrikulärem Thrombus wurden sowohl Fälle mit Niereninfarkt [8] als auch Schlaganfälle [5,6,9,10] beschrieben. In einer Arbeit von de Gregorio et al [3] traten in 5 von 15 Fällen mit linksventrikulärem Thrombus kardioembolische Ereignisse (davon 3 Schlaganfälle, 1 Niereninfarkt, 1 thrombotischer Poplitealarterienverschluss) auf.…”

Section: Abb 4 Koronarangiographie Mit Normaler Linkerunclassified

Neu aufgetretener linksventrikulärer Thrombus im Verlauf einer Tako-Tsubo-Kardiomyopathie

Michels

Pfister

2012

Dtsch med Wochenschr

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Takotsubo Syndrome: Clinical Features, Pathogenesis, Treatment, and Relationship with Cerebrovascular Diseases

Ranieri¹,

Finsterer

Bedini³

et al. 2018

Curr Neurol Neurosci Rep

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TTS is an increasingly recognized non-ischemic cardiomyopathy characterized by sudden, temporary weakening of the myocardium, of which the pathogenesis is unknown. Although pathogenesis of TTS remains unclear, a complex interaction between catecholamine-mediated stimulation, myocardial stunning, and subsequent stress-related myocardial dysfunction seems to be the main pathophysiological mechanism. Stroke is linked to TTS by a dual relationship since it may induce TTS by catecholamine release even if TTS itself also may be complicated by left ventricular thrombi leading to stroke. Given its possible complications, including the association with neurological diseases, both cardiologist and neurologists should be aware about TTS in order to diagnose it promptly and to initiate appropriate therapeutic measures.

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Apical thrombus in tako-tsubo cardiomyopathy

Cited by 4 publications

References 0 publications

Cardioembolic outcomes in stress-related cardiomyopathy complicated by ventricular thrombus: A systematic review of 26 clinical studies

Cardioembolic outcomes in stress-related cardiomyopathy complicated by ventricular thrombus: A systematic review of 26 clinical studies

Neu aufgetretener linksventrikulärer Thrombus im Verlauf einer Tako-Tsubo-Kardiomyopathie

Takotsubo Syndrome: Clinical Features, Pathogenesis, Treatment, and Relationship with Cerebrovascular Diseases

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