“…Chikungunya is described as an emerging and reemerging arbovirus transmitted to humans through the bite of Aedes mosquitoes, more precisely by females; The most well-known species are Aedes Aegypti, the most common vector in the Americas circulating in the territory since 1635, and Aedes Albopictus, which became important when it allowed better adaptation to the Chikungunya virus mutation, contributing to the expansion of the transmissibility of the disease in other parts of the world including Latin America. 1 Chikungunya virus is an RNA particle of the family Togaviridae and of the genus Alphavirus; It contains in its structure a genome that encodes structural (C, E1 and E2) and nonstructural (NsP1-4) proteins and its framework important glycoproteins for the recognition of organ and tissue cells; after infection, intracellular changes will be permanent with a single serotype, inducing the individual's immunization. [1][2] In 1952 the first human epidemic in East Africa was described; in southern Africa (Tanzania, Uganda and Zimbabwe) other cities were hit such as Bangkok, the Philippines, Cambodia, Vietnam, Laos, Myanmar, Malaysia and Indonesia successively until 1990, continuing to Thailand, Singapore and Sri Lanka.…”