Assessment of pulse wave velocity

Boutouyrie, Pierre; Briet, Marie; Collin, C.; Vermeersch, Sebastian; Pannier, Bruno

doi:10.1016/j.artres.2008.11.002

Cited by 104 publications

(91 citation statements)

References 40 publications

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“…This distension can be used to produce an optical signal which is correlated with the passing pressure wave. Several studies have shown that the distension and the pressure waveforms have an analogous wave contour and, therefore, can reciprocally be used for pulse wave analysis [9,28,47].…”

Section: Measurement Systemmentioning

confidence: 99%

An automatic method for arterial pulse waveform recognition using KNN and SVM classifiers

Pereira

Paiva

Correia

et al. 2015

Med Biol Eng Comput

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Section: Measurement Systemmentioning

confidence: 99%

An automatic method for arterial pulse waveform recognition using KNN and SVM classifiers

Pereira

Paiva

Correia

et al. 2015

Med Biol Eng Comput

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“…The pulse wave is generated by the contraction of the left ventricle, which causes initial shock pulse, counter the blood in the aorta. The pressure wave is recorded at these 2 sites (carotid and femoral) using sensors alternately on the carotid and the femoral site (42). Figure 4 shows the locations and the distance between the carotid and the femoral sites.…”

Section: The Carotid-femoral Pulse Wave Velocitymentioning

confidence: 99%

“…Aortic aging can be evaluated directly by measuring the PWV, using different devices. Pulse wave velocity, measured between carotid and femoral sites, is considered as the gold standard of aortic central aging (18); it is associated with age, hypertension, diabetes, and cholesterol, and also with kidney, heart failure, and cognitive impairment.…”

Section: Arterial Aging and Risk Factorsmentioning

confidence: 99%

Arterial Stiffness: A New Biomarker to be Measured

2017

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Arterial Stiffness (AS) is of great interest in the prediction and the management of cardiovascular events. The evaluation of AS using 2 points of measurement of the pulse wave velocity was of considerable increasing interest in the early 21st century. This paper aimed at focusing on the modeling of different measurement methods. Several measurement techniques are presented. A review of the bibliography and an explanation of the link between cardiovascular events and arterial stiffness are summarized. Novel apparatus that were developed for determining arterial stiffness claimed superiority over pioneering methods either through greater simplicity of use, better repeatability, or a more pertinent arterial pathway.

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“…[15][16][17][18][19][20][21] Although pathophysiologically very important, arterial stiffness is still a surrogate end point and what finally counts for the clinician (and for the patient) is the effect of treatment on cardiovascular morbidity and mortality (as proven in large randomized controlled trials). Therefore, there is still a need for data from prospective short term and long-term studies assessing the effects of the so-called 'cardiovascular protective drugs' on arterial aging and on clinical outcome data, thereby comparing results on the basis of central hemodynamic parameters with results based on peripheral measures.…”

mentioning

confidence: 99%

Cardiovascular prevention: relationships between arterial aging and chronic drug treatment

Backer

2011

J Hum Hypertens

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In hypertension, structural and functional properties of the arterial wall are altered, and morbidity and mortality associated with hypertension are related to the vascular damage affecting several organs.Going from central to peripheral arteries, the diameter and the composition of the vessel wall vary in important ways. In addition, the aorta and its main branches are highly sensitive to aging and changes in blood pressure while the more distal compartment is the major source of wave reflections and highly sensitive to vasoactive substances.Indeed, blood pressure propagation within the arterial tree is based on arterial stiffness, wave reflections and pulse pressure (PP). 1 Of note, arterial stiffness is an independent predictor for cardiovascular mortality and morbidity, and pulse wave velocity is currently accepted as the gold standard measure of arterial stiffness. [2][3][4] Early wave reflections, as evaluated by pulse wave analysis, are also an independent predictor of cardiovascular risk. 5 In the treatment of hypertension there is evidence by large randomized controlled trials and meta-analyses for a beneficial effect on hard endpoints (cardiovascular morbidity and mortality, stroke, kidney disease) for b-blockers, diuretics, calcium channel blockers, ACE inhibitors, angiotensin II receptor blockers. 6 In the first (but small) prospective randomised study, for example, Mackenzie et al. 7 investigated the effects of four major antihypertensive classes (atenolol, bendrofluazide, lercarnidipine, perindopril) on both central and peripheral blood pressures, PP amplification and aortic pulse wave velocity (PWV) in isolated systolic hypertension patients. They found that aortic PWV was not changed by any of the investigated drugs, and that despite similar reductions in peripheral systolic and pulse pressures with the four drugs, the changes in central pressure and augmentation index varied.Therapies combating arterial stiffness, reducing PP and reducing early wave reflections are believed to further reduce cardiovascular mortality. These treatments include exercise, nitrates, renin-angiotensin-aldosterone inhibitors, drugs acting on collagen cross-linking and vasopeptidase inhibitors. 8 Large scale, long term randomised controlled are necessary to prove that these agents effectively and safely diminish arterial stiffness and by this reduce cardiovascular morbidity and mortality.In the current issue of the Journal of Human Hypertension, Lieber et al. 9 report the results of a rather small cross-sectional analysis determining statistical associations between chronic drug treatment acting on cardiovascular prevention (antihypertensives, statins and oral antidiabetics and/or insulin) and age-induced changes in arterial stiffness and wave reflections. They conclude that calcium channel blockers and statins do not show any statistical association with PWV or wave reflection (augmentation index); that b-blockers are significantly associated with increased wave reflections and central PP; and that diuretics, angiotens...

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Assessment of pulse wave velocity

Cited by 104 publications

References 40 publications

An automatic method for arterial pulse waveform recognition using KNN and SVM classifiers

An automatic method for arterial pulse waveform recognition using KNN and SVM classifiers

Arterial Stiffness: A New Biomarker to be Measured

Cardiovascular prevention: relationships between arterial aging and chronic drug treatment

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