Author Correction: A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration

Silencing of microRNA-106b-5p prevents doxorubicin-mediated cardiotoxicity through modulation of the PR55α/YY1/sST2 signaling axis

“… 13 In this study, we focused on miRNA-106b-5p ( miR-106b ) because of its known role in regulating cardiac processes in human and mouse disease models. miR-106b is included in the miRNA106b-25 cluster 14 , 15 and, although its protective role in improving hypertrophy and cardiac function in left ventricular (LV) pressure overload and hypertrophy models has been reported, 16 , 17 , 18 it has also shown disparate results in other pathologies. In a study published in 2016, using an experimental animal model with cerebral ischemia/reperfusion injury, the authors demonstrated that its systemic silencing was able to ameliorate cerebral damage by blocking oxidative stress.…”