<b>Renal Artery Thrombosis Caused by Stent Fracture in a Single Kidney Patient</b>

Bessias, Nikolaos; Sfyroeras, George S.; Moulakakis, Konstantinos G.; Karakasis, Filippos; Ferentinou, Eleftheria; Andrikopoulos, Vasilios

doi:10.1583/05-1542.1

Cited by 27 publications

(18 citation statements)

References 16 publications

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“…Our results are consistent with those of a prior study and confirm the finding that kidney movement occurs with changes in body position (36). This movement is likely implicated in stent fracture (7)(8)(9)(10)(11). Additionally, inflammation and intimal hyperplasia caused by excessive stent movement may promote restenosis and thrombosis (17).…”

Section: Discussionsupporting

confidence: 92%

Evaluation of CT as a predictor for kidney and renal artery mobility

Guzzetta

Obara²,

Dachman³

2011

Diagn Interv Radiol

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Section: Discussionsupporting

confidence: 92%

Evaluation of CT as a predictor for kidney and renal artery mobility

Guzzetta

Obara²,

Dachman³

2011

Diagn Interv Radiol

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“…Recent reports on device failures in the coronary, 11 carotid, 12 renal, 2,16,36,38 aorta, 4,25,26 iliac, 37 popliteal, 46 and superficial femoral arteries (SFA), 1,18,39 have been a source of great concern in the medical device industry, among physicians and for regulatory agencies, stimulating research on the characterization of in vivo arterial deformation. A lack of knowledge of the in vivo biomechanical environment may lead to devices that are designed for deformations that are arguably much less important than others.…”

Section: Introductionmentioning

confidence: 99%

Methods for Quantifying Three-Dimensional Deformation of Arteries due to Pulsatile and Nonpulsatile Forces: Implications for the Design of Stents and Stent Grafts

Choi

Cheng

Wilson³

et al. 2008

Ann Biomed Eng

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The knowledge of dynamic changes in the vascular system has become increasingly important in ensuring the safety and efficacy of endovascular devices. We developed new methods for quantifying in vivo three-dimensional (3D) arterial deformation due to pulsatile and nonpulsatile forces. A two-dimensional threshold segmentation technique combined with a level set method enabled calculation of the consistent centroid of the cross-sectional vessel lumen, whereas an optimal Fourier smoothing technique was developed to eliminate spurious irregularities of the centerline connecting the centroids. Longitudinal strain and novel metrics for axial twist and curvature change were utilized to characterize 3D deformations of the abdominal aorta, common iliac artery, and superficial femoral artery (SFA) due to musculoskeletal motion and deformations of the coronary artery due to cardiac pulsatile motion. These illustrative applications show the significance of each deformation metric, revealing significant longitudinal strain and axial twist in the SFA and coronary artery, and pronounced changes in vessel curvature in the coronary artery and in the inferior region of the SFA. The proposed methods may aid in designing preclinical tests aimed at replicating dynamic in vivo conditions in the arterial tree for the purpose of developing more durable endovascular devices including stents and stent grafts.

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“…[15][16][17][18][19][20][21][22][23][24][25] Fractures in the femoropopliteal segment are associated with high restenosis rates and reduced patency. 21 Stent fracture has been reported to occur in areas with flexible arteries and in those with repetitive movements, such as the knee area.…”

Section: Discussionmentioning

confidence: 99%

Clinical relevance and treatment of carotid stent fractures

Sfyroeras

Koutsiaris

Karathanos

et al. 2010

Journal of Vascular Surgery

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Carotid stent fractures are mainly reported in self-expandable nitinol stents. Plaque calcification may be a risk factor for stent fractures. No difference was observed between open and closed-cell design. Stent fractures were often associated with restenosis and usually were asymptomatic. The actual incidence, clinical relevance, and optimal treatment remain to be clarified from larger prospective studies designed to investigate the issue.

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Renal Artery Thrombosis Caused by Stent Fracture in a Single Kidney Patient

Abstract: Incomplete expansion and fracture of the stent associated with insufficient antiplatelet therapy produced in-stent thrombosis. Collateral flow prevented kidney necrosis.

Cited by 27 publications

References 16 publications

Evaluation of CT as a predictor for kidney and renal artery mobility

Evaluation of CT as a predictor for kidney and renal artery mobility

Methods for Quantifying Three-Dimensional Deformation of Arteries due to Pulsatile and Nonpulsatile Forces: Implications for the Design of Stents and Stent Grafts

Clinical relevance and treatment of carotid stent fractures

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