Bartter's syndrome

Solomon, Richard; Brown, Robert S.

doi:10.1016/0002-9343(75)90265-x

Cited by 26 publications

(3 citation statements)

References 28 publications

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“…Bartter's description of a syndrome of secondary hyperaldosteronism with normal blood pressure was first made in 1962. Over the years a great deal of information has accumulated concerning the role of plasma renin, the pathologic status of the juxtaglomerular apparatus and the renal tubular handling of urinary anions (1, 3, 6, 7).…”

Section: Discussionmentioning

confidence: 99%

“…The efficacy of indomethacin, ibuprofen and aspirin has been confirmed. These agents appear to be far more effective than spironolactone, propranolol or potassium supplementation in reversing the hypokalemia (1, 4, 6).…”

Section: Discussionmentioning

confidence: 99%

“…The etiology of Bartter's syndrome is not known completely. Previous papers have shown that hyperreninemia and secondary hyperaldosteronism are involved in the renal potassium loss which results in hypokalemia (1–3). Recently it has been demonstrated that suppression of prostaglandin synthesis can reverse the hypokalemia (4, 5).…”

mentioning

confidence: 99%

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Bartter's Syndrome in a 74‐Year‐Old Woman: Response to Anti‐Inflammatory Agents

Gordon

Pollard

1980

J American Geriatrics Society

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The occurrence of Bartter's syndrome is described in a 74-year-old woman with a fractured hip. She showed hypokalemia and elevated levels of plasma renin and aldosterone. Only with difficulty did large doses of potassium supplementation raise the serum potassium level. After discontinuation of the exogenous potassium, there was a good response to treatment with anti-inflammatory agents such as indomethacin and aspirin.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Bartter's Syndrome in a 74‐Year‐Old Woman: Response to Anti‐Inflammatory Agents

Gordon

Pollard

1980

J American Geriatrics Society

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Blood and Extracellular Fluid Volume in Patients With Bartter's Syndrome

Boer¹

1983

Arch Intern Med

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Treatment of Bartter's Syndrome With Indomethacin

Garin¹

1980

Arch Pediatr Adolesc Med

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\s=b\Two patients with Bartter's syndrome were treated with indomethacin (2 mg/ kg/day). The administration of the drug resulted in weight gain; a decrease in the rate of urinary excretion of sodium and inorganic phosphate suggesting an increase in proximal tubular reabsorption; an increase in serum potassium concentration, with a transient decrease in the rate of urinary potassium excretion in one patient; and a decrease in plasma renin activity and in the rate of urinary aldosterone excretion. Since indomethacin has been shown to inhibit prostaglandin synthetase, these observations support the hypothesis that prostaglandin excess is a basic pathogenic mechanism in Bartter's syndrome. (Am J Dis Child 134:258-261, 1980)

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Bartter's syndrome

Cited by 26 publications

References 28 publications

Bartter's Syndrome in a 74‐Year‐Old Woman: Response to Anti‐Inflammatory Agents

Bartter's Syndrome in a 74‐Year‐Old Woman: Response to Anti‐Inflammatory Agents

Blood and Extracellular Fluid Volume in Patients With Bartter's Syndrome

Treatment of Bartter's Syndrome With Indomethacin

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