�ber Temperatur- und Kreislaufzeitmessungen in verschiedenen Abschnitten der Hundeniere

Ochwadt, Bruno; Schmier, J

doi:10.1007/bf00363634

Cited by 15 publications

(5 citation statements)

References 11 publications

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“…This is in accordance with data obtained by the thermal dilution method (Ochwadt & Schmier, 1954;Schieve et al, 1959). On the other hand, Auckland, Bower & Berliner (1964) found with the hydrogen gas method that the blood flow was very much the same in the outer, middle and inner third of cortex, and similarly Wolgast (1968) showed with the 32P method that in cortex the blood flow was nearly the same at all distances from the cortico-medullary junction.…”

Section: Distribution Of Bloodflow In the Renal Cortexsupporting

confidence: 93%

Distribution of Blood Flow in the Renal Cortex during Saline Loading

1970

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1. The effect of infusion of isotonic saline on the circulation in the renal cortex in the dog was investigated by an external counting technique involving measurement of the transit times for 'Krypton and l 311-labelled albumin after rapid injection into the renal artery.2. During saline infusion superficial renal cortical blood flow and overall cortical blood flow rose by 23 and 15%, respectively. There was a 6% rise in the ratio superficial cortical blood flow to overall cortical flow, which however, was not significant.3. Resistance to flow through cortex decreased. 4. Mean transit time for plasma through cortex decreased from an average of 2.9 sec in the control period to 2.6 and 2.1 sec during saline infusion. 5.Renal cortical blood volume, as estimated from the cortical blood flow and the mean transit time for plasma, was virtually unchanged.6. These studies indicate that the decrease in resistance to flow during acute isotonic saline infusion is probably caused by a dilatation of the resistance vessels only. No significant redistribution of blood flow in cortex takes place; this, however, does not exclude regional changes in glomerular filtration rate. Physiologie, 284, 195-200. LASSEN, N.A. & MUNCK, 0. (1955) The cerebral blood flow in man determined by the use of radioactive krypton. Acta Physiologica Scandinavica, 33, 3049. 44,929-941. Academy of Sciences, 139, 312-327. medullary glomeruli in the rat kidney. Pfliigers Archiv fur die gesamte Physiologie, 301, 162-181. G

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Section: Distribution Of Bloodflow In the Renal Cortexsupporting

confidence: 93%

Distribution of Blood Flow in the Renal Cortex during Saline Loading

1970

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“…On the other hand, Sadler and Tuttle (1963) found more pronounced reduction of cortical than of medullary heat clearance during noradrenaline infusion, but since medullary heat clearance is to a great extent determined by diffusion and not by blood flow (Aukland 1967b), the clearances cannot be directly interpreted in terms of local blood flow. This difficulty with heat as indicator may possibly also explain the finding of a greater prolongation of cortical than of medullary heat appearance time during adrenaline infusion (Ochwadt and Schmier 1954).…”

Section: Discussionmentioning

confidence: 99%

“…Among the numerous investigators repeating Trueta's experiments with similar methods, some confirmed and others denied the existence of the "Trueta shunt", and several authors emphasised the difficulties of producing a medullary diversion in the dog. More quantitative methods, such as local heat clearance (Scher 1951, Sadler andTuttle 1963), heat appearance time (Ochwadt and Schmier 1954), PAR extraction (Reubi and Schroeder 1949), and externally recorded Krypton'" clearance (Carriere et at. 1966) have also given conflicting results.…”

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confidence: 99%

Effect of Adrenaline, Noradrenaline, Angiotensin and Renal Nerve Stimulation on Intrarenal Distribution of Blood Flow in Dogs

Aukland

1968

Acta Physiologica Scandinavica

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_ AUKLAND, K. Effect of adrenaline, noradrenaline, angiotension and renal nerve stimulation on intrarenal distribution of blood flow in dogs. Acta physiol. scand. 1968. 72. 498-509. The acute effect of adrenaline, noradrenaline, angiotensin and renal nerve stimulation on intrarenal blood flow was studied in 15 dogs. Local clearance of hydrogen gas measured polarographically with 2--4 needle-shaped platinum electrodes in the outer medulla served as indicator for medullary blood flow. Total renal blood flow was measured simultaneously with electromagnetic flowmeter. Adrenaline, noradrenaline and angiotensin infused i.v. or into the renal artery over a period of 5-15 min reduced outer medullary hydrogen clearance and total renal blood flow to 25-90 per cent of control, and on an average to the same extent. Renal nerve stimulation produced the same pattern with flow reduction to 50--75 per cent of control. Since changes in total renal blood flow mainly reflect changes in cortical perfusion, it is concluded that resistance to flow was increased proportionately in the cortical and juxtamedullary circulation. Considerable local variations in response of outer medullary hydrogen clearance observed with all stimuli suggested local differences in vasoconstrictor response, but no consistent difference between cortex and medulla.The acute effect of adrenaline, noradrenaline, angiotensin and renal nerve stimulation is to increase renal vascular resistance. In dogs, angiotensin and renal nerve stimulation may reduce blood flow to 25-50 per cent of control, whereas catecholamine injections may cause complete cessation of renal blood flow. In the latter case, it is obvious that the whole kidney becomes ischemic, but it is conceivable that smaller doses could produce selective ischemia in parts of the kidney, or at least a change in the normal intrarenal flow distribution. Such a response type was proposed by Trueta et al. (1947) who reported that adrenaline infusion, crush injuries and nerve stimulation in rabbits often caused complete cortical ischemia, while the blood was diverted through the juxtamedullary circulation. The same response was later observed after renin injection (Daniel, Prichard and Ward-McQuaid 1954). This conclusion was based on qualitative methods, such as India ink injec-498

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“…a. ) noch von physiologischer Seite (Maxwell et al, 1950;Lamport, 1950;Smith, Homer W., 1951;Rothlin und Cerletti, 1952;Piiper und Schürmeyer, 1955;Ochwadt, 1954Ochwadt, , 1956 -bestätigt worden, so daß die Frage nach der Existenz quantitativ ins Gewicht fallender arteriovenöser Anastomosen in der Niere heute verneinend beantwortet werden muß. Wenig befriedigend ist es indessen, daß man bislang keine ausreichende Erklärung dafür hatte, daß ein so erfahrener Untersucher wie Spanner mehr als 250 ,,arterio-venöse Anastomosen" auf dem qcm im Bereich des Pelvis renalis und der Capsula fibrosa finden konnte.…”

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“…In aller Kürze seien die hier gebräuchlichen Begriffe einander gegenübergestellt. Zweifach sowie Chambers und Zweifach et al (1936/37, 1937, 1939a, b, 1945/46, 1947/48 a, b, 1948, 1954 beschreiben im Bereich der terminalen Strombahn eine funktionelle Einheit", die sich u. a. aus folgenden Abschnitten zusammensetzt: L ,,a-v channel" ( ,,thoroughfare-" = preferential-" = ,,central channel'). Dieser verbindet Endarteriolen mit Venulen und zeigt in seinem proximalen Abschnitt ( ,,metarteriole") typische", aber diskontinuierlich verteilte glatte Muskeizellen, während der distale Abschnitt zunehmend muskelärmer und als distales Segment" oder ,,a-v capillary" bezeichnet wird.…”

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Gibt es arterio-venöse Anastomosen in der Niere?

Hammersen¹

1962

Dtsch med Wochenschr

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�ber Temperatur- und Kreislaufzeitmessungen in verschiedenen Abschnitten der Hundeniere

Cited by 15 publications

References 11 publications

Distribution of Blood Flow in the Renal Cortex during Saline Loading

Distribution of Blood Flow in the Renal Cortex during Saline Loading

Effect of Adrenaline, Noradrenaline, Angiotensin and Renal Nerve Stimulation on Intrarenal Distribution of Blood Flow in Dogs

Gibt es arterio-venöse Anastomosen in der Niere?

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