2016
DOI: 10.4103/0028-3886.173671
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Bilateral lateral geniculate body hemorrhagic infarction: A rare cause of acute bilateral painless vision loss in female patients

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Cited by 5 publications
(5 citation statements)
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“…There was no documented hyponatremia, so possibility of osmotic demyelination could be excluded. The literature similar to our case of acute onset painless vision loss due to coagulative necrosis of LGB induced by acute pancreatitis with microangiopathic hemolytic anemia was described by Mathew et al [6] and Mudumbai and Bhandari. [3]…”
Section: Name Of the Study Chracteristics Of Study Population Possibl...supporting
confidence: 84%
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“…There was no documented hyponatremia, so possibility of osmotic demyelination could be excluded. The literature similar to our case of acute onset painless vision loss due to coagulative necrosis of LGB induced by acute pancreatitis with microangiopathic hemolytic anemia was described by Mathew et al [6] and Mudumbai and Bhandari. [3]…”
Section: Name Of the Study Chracteristics Of Study Population Possibl...supporting
confidence: 84%
“…LGB infarction with Inflammatory bowel disease and recurrent pancreatitis. Mathew T et al [6] 2016 42/F Vascular occlusion due to microangiopathic disorders cortices. [4] Isolated LGB involvement causing bilateral vision loss is rare with very few literature available on the topic.…”
Section: Name Of the Study Chracteristics Of Study Population Possibl...mentioning
confidence: 99%
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“…The mechanisms causing damage to LGBs are as follows: infarction, myelinolysis, or hypoperfusion with predilection to young females. [23] None of these were associated with pregnancy and influenza infection as in our case.…”
mentioning
confidence: 46%
“…Since the 1930s, there are only nine case reports in the literature of lesions in LGBs, resulting in bilateral acute painless vision loss. [23] The different etiologies for bilateral lesions described include the following: pancreatitis, cirrhosis with hyponatremia, febrile gastroenteritis, preeclampsia, anaphylactic shock, and stroke. The mechanisms causing damage to LGBs are as follows: infarction, myelinolysis, or hypoperfusion with predilection to young females.…”
mentioning
confidence: 99%