Biometrical genetic models of self-reported delinquent behavior: A twin study

Rowe, David C.

doi:10.1007/bf01065923

Cited by 190 publications

(108 citation statements)

References 13 publications

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“…An alternative source of shared environmental influences found that using twin designs may be sibling effects, or the effects of shared peers. In one of the first twin studies of adolescent delinquency, Rowe (1983) reported that a substantial proportion of same-age, same-sex twin pairs reported co-offending (i.e. participating in delinquent activities together).…”

Section: Introductionmentioning

confidence: 99%

A genetically informative developmental study of the relationship between conduct disorder and peer deviance in males

et al. 2007

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Section: Introductionmentioning

confidence: 99%

A genetically informative developmental study of the relationship between conduct disorder and peer deviance in males

et al. 2007

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“…Whereas some studies have found evidence for substantial genetic influences in the development of childhood antisocial behavior problems (Eaves et al, 1997;Grove et al, 1990;Rowe, 1983;Slutske et al, 1997;1\vito & Stewart, 1982), others have not (Lyons et al, 1995; see also Cloninger & Gottesman, 1987;DiLalla & Gottesman, 1989). For example, in two recent twin studies of adult-reported childhood CD (the largest twin studies of CD to date), the estimates of the contribution of genetic factors to variation in risk for CD were 7% (Lyons et al, 1995) versus 43%-71 % .…”

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Common genetic risk factors for conduct disorder and alcohol dependence.

Slutske

Heath

Dinwiddie

et al. 1998

Journal of Abnormal Psychology

275

225

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The association between retrospectively reported childhood conduct disorder (CD) and a history of alcohol dependence (AD) was examined in a sample of 2,682 male, female, and unlike-sex adult twin pairs. There was a strong association between CD and AD in both men (tetrachoric r = .34, odds ratio = 2.8) and women (tetrachoric r = .53, odds ratio = 9.9). Genetic factors accounted for most of the association between CD and AD liability in men and women, with the remainder of the association being due to nonshared individual-specific environmental factors. Genetic inftuences common to CD and AD accounted for 17% and 35% of the genetic variation in AD liability in men and women, respectively, and accounted for 11 % and 23% of the total variation in AD liability in men and women, respectively. The results suggest that there are common genetic risk factors for CD and AD or that CD itself is an important genetically influenced risk factor for AD.

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“…1989; Raine, 1993;Rutter et al, 1990). although some studies have found evidence of substantial genetic infiuences (Cadoret. Yates, 'Irougbton, Woodworth, & Stewart, 1995;Eaves et aI.. 1995;Grove et al, 1990;Rowe, 1983;Twito & Stewart. 1982;Waldman, Pickens, & Svikis, 1989).…”

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confidence: 99%

“…In addition, most recent twin studies of childhood antisocial behaviors (e.g., Edelbrock, Rende, Plomin, & Thompson, 1995;Grove et al, 1990;Lyons, et Rowe, 1983) have used continuous measures such as behavior or symptom counts rather than CD diagnoses. The inhc:ri.tance of antisocial or delinquent symptoms may or may not be the same as the inheritance of the more extreme pattern of behavior that couesponds to diagnosable CD.…”

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confidence: 99%

“…Lyons et aL, 1995;'1Wito & Stewart. 1982) or unlike-sex sibling or twin pairs (e.g., Edelbrock et Rowe, 1983;Stevenson & Oraham, 1988) were not available. Some SbJdies suggest that genetic infiuences may account for a larger proportion of the variation in childhood antisocial behaviors in boys than in girls (Silberg et aI., 1994;Stevenson & Graham, 1988;van den Oord, Boomsma, & Verhnlst, 1994), although this is not universally found (Rowe, 1983) and is inconsistent with the lituature on childhood antisocial behavior in boys (including the VietDam-era twin study)…”

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confidence: 99%

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Modeling genetic and environmental influences in the etiology of conduct disorder: A study of 2,682 adult twin pairs.

Slutske¹,

Heath²,

Dinwiddie³

et al. 1997

Journal of Abnormal Psychology

183

168

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The etiology of CODdw:t disorder (CD) was examined retrospectively in a sample of 2,682 male.female, and un1ike-sex adult twin pairs from the community·based Australian 1Win Registec Modelfitting analyses indicated a substantial genetic intluence on risk for CD, accountiDg for 71<11 of the variance (95% confidence interval [CI] = 32-79<11). There was DOt a statistically sigui1icam effect of the shared environment in the best-fitting model of CD, but a modest effect of the shared environment on the risk for CD could not be rejected (95% CI = 0-32%). 1bemagoitude of genetic aDd environmental intluences for CD liability did not vary significantly for boys aDd girls. and the specific genetic aDd enviromnenta1 mechanisms important for the developmeat of CD appeared to be largely the san1e for both sexes. The fit of a multiple-thresbold model raises the possibility that CD may not necessarily be a discrete entity but rather an extreme of the normal variation in CODductdisordered behavior found in the general population.

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Biometrical genetic models of self-reported delinquent behavior: A twin study

Cited by 190 publications

References 13 publications

A genetically informative developmental study of the relationship between conduct disorder and peer deviance in males

A genetically informative developmental study of the relationship between conduct disorder and peer deviance in males

Common genetic risk factors for conduct disorder and alcohol dependence.

Modeling genetic and environmental influences in the etiology of conduct disorder: A study of 2,682 adult twin pairs.

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