Chiasmal optic neuritis following mumps parotitis

Irioka, Takashi; Akaza, Miho; Nakao, Keisuke; Kanouchi, Tadashi; Yokota, Takanori; Mizusawa, Hidehiro

doi:10.1007/s00415-008-0810-1

Cited by 10 publications

(8 citation statements)

References 13 publications

(18 reference statements)

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“…Interestingly, a case report of a 10-year-old boy developing mumps during acute myelitis also described a very long spinal cord lesion (from C3 to T12) on MRI, which is characteristic of NMO, although anti-AQP4 antibody was not mentioned [22]. Moreover, the optic chiasma was primarily perturbed in a case of acute optic neuritis following mumps virus infection [23]. Although this paper did not mention the presence of anti-AQP4 antibody, this clinical picture suggests that this patient also had an anti-AQP4 antibody-related disorder [24].…”

Section: Discussionmentioning

confidence: 99%

A serological analysis of viral and bacterial infections associated with neuromyelitis optica

Koga

Takahashi

Kawai

et al. 2011

Journal of the Neurological Sciences

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Section: Discussionmentioning

confidence: 99%

A serological analysis of viral and bacterial infections associated with neuromyelitis optica

Koga

Takahashi

Kawai

et al. 2011

Journal of the Neurological Sciences

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“…On the other hand, chiasmal involvement may be induced by extrinsic or intrinsic abnormalities [3,4]. Regarding intrinsic chiasmal lesions, neoplasm [3,[5][6][7], chiasmal infarction [3,8,9], sarcoidosis [3,10], and OCN [1,3,[11][12][13][14][15][16][17][18][19] have been reported. Among them, OCN is a relatively rare symptom consisting of acute visual loss due to chiasmal infl ammation [1,3,[11][12][13][14][15][16][17][18][19], and commonly occurs as an idiopathic event, either in isolation or in the setting of multiple sclerosis [3,11].…”

Section: Discussionmentioning

confidence: 99%

“…Regarding intrinsic chiasmal lesions, neoplasm [3,[5][6][7], chiasmal infarction [3,8,9], sarcoidosis [3,10], and OCN [1,3,[11][12][13][14][15][16][17][18][19] have been reported. Among them, OCN is a relatively rare symptom consisting of acute visual loss due to chiasmal infl ammation [1,3,[11][12][13][14][15][16][17][18][19], and commonly occurs as an idiopathic event, either in isolation or in the setting of multiple sclerosis [3,11]. Furthermore, previous reports have noted that VZV infection, infectious mononucleosis due to EBV [12], CMV infection [13], mumps parotitis [14], Lyme disease [15], neuromyelitis optica [16], systemic lupus erythematosus [17], vitamin B12 defi ciency [18], and ethchlorvynol toxicity [19] might induce OCN.…”

Section: Discussionmentioning

confidence: 99%

Isolated Optic Chiasmal Neuritis Secondary to Varicella-Zoster Virus Infection

Tsuda

2011

J Med Cases

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A 49-year-old woman without contributory medical history presented with zoster in the left-sided second cervical dermatome, and underwent oral valaciclovir at 3000 mg/day. Five days later, bitemporal hemianopia developed. Corrected visual acuities were spared, and funduscopic fi ndings demonstrated no abnormalities in both eyes. Varicella-zoster virus (VZV) viremia was proved by the polymerase chain reaction test. Cerebrospinal fl uid examination fi ndings were normal. Cranial and orbital magnetic resonance imaging demonstrated no abnormalities. Thereafter, under 7 days of intravenous aciclovir at 1500 mg/day, bitemporal hemianopia gradually improved. This is a fi rst reported case of isolated optic chiasmal neuritis secondary to VZV infection.

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“…Tuberculosis, Lyme disease, cryptococcus, cysticercosis, allergic fungal rhinosinusitis, aspergillosis, cytomegalovirus, mumps, and Epstein-Barr virus have all been associated with chiasmal inflammation. [43][44][45] As with all infectious diseases, treatment should be directed at the underlying cause.…”

Section: Inflammatorymentioning

confidence: 99%

Visual Findings in Chiasmal Syndromes

Foroozan¹

2016

International Ophthalmology Clinics

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The optic chiasm is an important neuroanatomic landmark, in particular as it relates to the pituitary gland and endocrinologic syndromes. Pathology that affects the chiasm helps emphasize key principles of neuroanatomy. The most common visual consequence of pituitary tumors is chiasmal compression, and visual findings remain important indicators of central nervous system pathology in many patients with sellar/suprasellar lesions.In this review, I have illustrated several different clinical syndromes that ensue from pathology involving the optic chiasm and discussed the various etiologies of chiasmal pathology. In addition, I have provided several examples of the neuroimaging findings that highlight the anatomy and pathology involving the optic chiasm.I hoped to provide a logical basis for the practicing ophthalmologist in approaching chiasmal disorders. ' AnatomyThe intracranial optic nerves exit the optic canals and travel at a 30-to 45-degree angle to make up the optic chiasm. The optic chiasm is 12 to 18 mm in diameter and contains the decussation of nasal retinal nerve fibers from each of the optic nerves. 1 Inferior retinal fibers representing the superior visual field cross first, whereas macular fibers cross more posteriorly. The chiasm lies 10 mm above the sella turcica, which contains the pituitary gland (Fig. 1).The space inferior to the chiasm and above the sella is the basal cistern. Anteriorly, the chiasm is exposed to cerebrospinal fluid; posteriorly it abuts the hypothalamus and the anterior portion of the third ventricle. The anatomic position of the chiasm is described as

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Chiasmal optic neuritis following mumps parotitis

Cited by 10 publications

References 13 publications

A serological analysis of viral and bacterial infections associated with neuromyelitis optica

A serological analysis of viral and bacterial infections associated with neuromyelitis optica

Isolated Optic Chiasmal Neuritis Secondary to Varicella-Zoster Virus Infection

Visual Findings in Chiasmal Syndromes

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