Childhood absence epilepsy: Elctroclinical features and diagnostic criteria

Ma, Xiuwei; Zhang, Yuehua; Yang, Zhixian; Liu, Xiaoyan; Sun, Huihui; Qin, Jiong; Wu, Xiru; Liang, Jing

doi:10.1016/j.braindev.2010.02.004

Cited by 27 publications

(27 citation statements)

References 13 publications

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“…In none was a typical event captured confirming a diagnosis of epilepsy. This is in striking contrast to children with typical childhood absence epilepsy, in whom clinical absences with generalised 3 Hz spike-wave are commonly recorded during routine EEG 12. Focal dyscognitive seizures, although more commonly seen in children with ASD compared with the normal paediatric population,13 14 are not usually recorded during routine EEG.…”

Section: Discussionmentioning

confidence: 76%

Limited role for routine EEG in the assessment of staring in children with autism spectrum disorder

2014

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Section: Discussionmentioning

confidence: 76%

Limited role for routine EEG in the assessment of staring in children with autism spectrum disorder

2014

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“…Absence seizures are brief, generalized epileptic seizures of sudden onset and termination. Impairment of consciousness and generalized spike-and-slow wave discharges (SWD) on EEG are two essential feature of absence seizure (1). …”

Section: Introductionmentioning

confidence: 99%

The Role of T-Type Calcium Channel Genes in Absence Seizures

2014

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The thalamic relay neurons, reticular thalamic nucleus, and neocortical pyramidal cells form a circuit that sustains oscillatory burst firing, and is regarded as the underlying mechanism of absence seizures. T-type calcium channels play a key role in this circuit. Here, we review the role of T-type calcium channel genes in the development of absence seizures, and emphasize gain or loss of function mutations, and other variations that alter both quantity and quality of transcripts, and methylation status of isoforms of T-type calcium channel proteins might be of equal importance in understanding the pathological mechanism of absence seizures.

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“…The seizure can last for 3 s to as long as 40 s. Absences occur spontaneously but are also readily provoked by hyperventilation and occasionally by photic stimulation. The electroencephalography (EEG) reveals bilateral, synchronous symmetrical 3 Hz spike-and-slow-wave-discharge (SWD) on a normal background activity[2]–[4].…”

Section: Introductionmentioning

confidence: 99%

Localization of epileptic foci in Children with childhood absence epilepsy by magnetoencephalography combined with synthetic aperture magnetometry

Dong

Wang

et al. 2011

Journal of Biomedical Research

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This present study was aimed to investigate the localizable diagnostic value of magnetoencephalography (MEG) combined with synthetic aperture magnetometry (SAM) in childhood absence epilepsy (CAE). Thirteen CAE patients underwent MEG detection at resting state and after hyperventilation, and then the epileptic foci were located by SAM. In the thirteen CAE patients, epileptic foci were found in five cases (38.5%), and they were all located in the bilateral frontal lobe, suggesting that the frontal lobe in some CAE patients may serve as the epileptic foci. Our findings indicate that MEG combined with SAM could be of diagnostic value in localizing the epileptic foci in certain CAE patients.

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Childhood absence epilepsy: Elctroclinical features and diagnostic criteria

Cited by 27 publications

References 13 publications

Limited role for routine EEG in the assessment of staring in children with autism spectrum disorder

Limited role for routine EEG in the assessment of staring in children with autism spectrum disorder

The Role of T-Type Calcium Channel Genes in Absence Seizures

Localization of epileptic foci in Children with childhood absence epilepsy by magnetoencephalography combined with synthetic aperture magnetometry

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