Chronic carbon monoxide poisoning in children

Khan, Kori; Sharief, N.

doi:10.1111/j.1651-2227.1995.tb13747.x

Cited by 9 publications

(3 citation statements)

References 3 publications

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“…However, all these reports have uncontrolled confounding factors and lack data regarding the exposure [20,[144][145][146][147][148][149][150]. These symptoms typically abate once the patient is removed from the environment [19,20,145,148,151].…”

Section: Clinical Effects: Chronicmentioning

confidence: 98%

Carbon Monoxide Poisoning

Kao

Nañagas

2005

Medical Clinics of North America

100

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Section: Clinical Effects: Chronicmentioning

confidence: 98%

Carbon Monoxide Poisoning

Kao

Nañagas

2005

Medical Clinics of North America

100

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“…Chronic exposure to CO has also been associated with decreased physical performance, exacerbation of heart disease, and low birth weight. Furthermore, chronic hypoxia determines polycythemia and cardiomegaly [136][137][138][139][140][141].…”

Section: Chronic Clinical Effectsmentioning

confidence: 99%

“…The risk factors include an age over 40 years, cardiovascular disease, prolonged exposure to CO (greater than 1 h), electroencephalographic abnormalities, and coma (especially if prolonged and followed by persistent asthenia and vertigo). It has been observed that patients with a broader symptomatic clinical picture at onset have a greater chance of developing long-term neurological sequelae [140,[145][146][147].…”

Section: Delayed Neurological Syndrome (Dns)mentioning

confidence: 99%

Carbon Monoxide Poisoning: From Occupational Health to Emergency Medicine

Savioli,

Gri,

Ceresa

et al. 2024

JCM

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Carbon monoxide poisoning remains a leading cause of accidental poisoning worldwide (both at home and at work), and it is also a cause of suicidal poisoning. Such poisoning can arise following prolonged exposure to low levels of CO or following brief exposure to high concentrations of the gas. In fact, despite exposure limits, high safety standards, and the availability of CO alarms, nearly 50,000 people in the United States visit the emergency department each year due to poisoning. Additionally, CO poisoning in the United States causes up to 500 deaths each year. Despite the widespread nature of this form of poisoning, known about for centuries and whose damage mechanisms have been recognized (or rather hypothesized about) since the 1800s, early recognition, especially of late complications, and treatment remain a medical challenge. A well-designed therapeutic diagnostic process is necessary so that indication for hyperbaric or normobaric therapy is correctly made and so that patients are followed up even after acute exposure to diagnose late complications early. Furthermore, it is necessary to consider that in the setting of emergency medicine, CO poisoning can be part of a differential diagnosis along with other more frequent conditions, making its recognition difficult. The last thirty years have been marked by a significant increase in knowledge regarding the toxicity of CO, as well as its functioning and its importance at physiological concentrations in mammalian systems. This review, taking into account the significant progress made in recent years, aims to reconsider the pathogenicity of CO, which is not trivially just poisonous to tissues. A revision of the paradigm, especially as regards treatment and sequelae, appears necessary, and new studies should focus on this new point of view.

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Frontal Lobe Disorders in Pediatric Neuropsychology: Attention-Deficit Hyperactivity Disorder and Tourette Disorder

Rostain

2010

Handbook of Medical Neuropsychology

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Chronic carbon monoxide poisoning in children

Cited by 9 publications

References 3 publications

Carbon Monoxide Poisoning

Carbon Monoxide Poisoning

Carbon Monoxide Poisoning: From Occupational Health to Emergency Medicine

Frontal Lobe Disorders in Pediatric Neuropsychology: Attention-Deficit Hyperactivity Disorder and Tourette Disorder

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