2015
DOI: 10.1016/j.cobeha.2015.05.003
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Cognitive reserve and neuropsychiatric disorders

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Cited by 13 publications
(11 citation statements)
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“…Previous literature has been focused on the reserve effect in cognitive tasks. Our study further suggests that education plays role in broad psychosocial functioning ( Barnett et al, 2006 , Watson and Joyce, 2015 ). In this study, the mood dysregulation component of NPS was associated with thinner cortices in the left inferior and orbitofrontal cortex.…”
Section: Discussionsupporting
confidence: 67%
“…Previous literature has been focused on the reserve effect in cognitive tasks. Our study further suggests that education plays role in broad psychosocial functioning ( Barnett et al, 2006 , Watson and Joyce, 2015 ). In this study, the mood dysregulation component of NPS was associated with thinner cortices in the left inferior and orbitofrontal cortex.…”
Section: Discussionsupporting
confidence: 67%
“…On the basis of these findings, it is plausible that differential cognitive performance across subgroups is a function of cognitive reserve for the intact, the intermediate impairment and the severe impairment subgroups. This is consistent with the theoretical concept of cognitive reserve in dementia and schizophrenia spectrum or mood disorders, as a protective mechanism against cognitive decline caused by neuropathology itself 13 , 38 or against the adverse cognitive effects of certain treatments (e.g. memory performance after electroconvulsive therapy).…”
Section: Discussionsupporting
confidence: 88%
“…Subjects with lower education attainment had higher hyper-connectivity and worse hypo-connectivity, reflecting that they may have lower neural resources and that the presence of hyper-connectivity and hypo-connectivity are reciprocal dynamic changes. Education attainment is often deemed as the proxy measure for cognitive reserve, and subjects with higher cognitive reserve could tolerate more brain pathology and bypass the needs to recruit compensatory resources ( Steffener and Stern, 2012 ; Watson and Joyce, 2015 ). Furthermore, the correlations between education attainment and connectivity alterations may suggest that the observed hyper-connectivity and hypo-connectivity in CS patients is related to neuronal activity and not simply the blood flow effect.…”
Section: Discussionmentioning
confidence: 99%