Comparative effects of four herbicides on physiological aspects in Triticum sativum L.

Abstract

Four herbicide (topik, puma super, granstar and derby) treatments at the recommended field dose (RFD) were applied for 5 weeks on wheat (Triticum sativum L. cv. Giza 168) crop. These selected herbicides are recommended to control two types (narrow and broad) of weeds in this crop. The growth parameter, oxidative stress defence system, nitrate reduction, proline level and protein contents were used to evaluate the toxic effect of these herbicides on wheat crop. The toxicity of herbicides used on this crop was as follows: puma super > topik > derby > granstar. The correlation between the herbicide concentrations and the activities of the antioxidant enzymes was monitored.

Potassium Synergize the Positive Effect of Ascorbic Acid on some Morpho-physiological Parameters of Salt Stressed Faba Bean Cultivars

“…b content of Giza 716 (41%) was higher than that of Giza 843. The negative effects of salinity on photosynthetic pigments were described in other reports (El-Tayeb, 2005;Faheed, 2012;AbdElgawad et al, 2016;Wang et al, 2017).…”

“…The highest values of K were detected when 15mM K was used. High concentration of NaCl in growth medium reduced the uptake of K + ions (Alpaslan & Gunes, 2001;Faheed, 2012;Noreen et al, 2017), it was reversed when sufficient K content of nutrient solution (15mM K) was used, and it was synergized in AsA treated plants.…”

Toxicological Effects of a Post Emergent Herbicide on Spirodela polyrhiza as a Model Macrophyte: A Comparison of the Effects of Pure and Nano-capsulated Form of the Herbicide

“…A similar trend has been previously reported in the case of the plant species treated with aryloxyphenoxy propionate Volume 12, No 2, March-April 2018; http://www.ijt.ir group of herbicides such as Quizalofop-p-ethyl and fluazifop-p-buthyl [10,11,30]. This group of herbicides can indirectly disrupt fatty acid biosynthesis via the inhibition of ACCase [11,31,32].…”

Short-term phytotoxicity in Brassica napus (L.) in response to pre-emergently applied metazachlor: A microcosm study

“…That these responses in membrane destabilization became significant after 4 wk could be explained by either the fact that the inhibition of very long chain fatty acids is a relatively slow process [23] or the fact that oxidative damage is a secondary side effect of metazachlor. In general, herbicide-induced oxidative stress has been described in several crops [18,19], in particular for chloroacetamides such as alachlor and metolachlor [20,21]. The induction of the antioxidative enzymes SOD, AP X , CAT, and GR at 2 wk and 4 wk after metazachlor application indicated metazachlor-induced oxidative stress (Tables 1 and 2).…”

“…Initially after herbicide application, when detoxification is either activated or at its full turnover, the nondetoxified fraction of cytosolic herbicidal compounds can indirectly cause oxidative damage at different cellular levels by the induction of oxidative stress. Herbicide‐induced oxidative stress has been described in crops and for chloroacetamides, such as alachlor and metolachlor, in particular . Herbicides from different classes of mode of action can negatively affect crop morphology and physiology, ranging from destabilization of cellular membranes to pigment profiles .…”