2022
DOI: 10.1016/j.nut.2022.111588
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Comparison of the effects of different dietary regimens on susceptibility to experimental acute kidney injury: The roles of SIRT1 and TGF-β1

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Cited by 10 publications
(9 citation statements)
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“…The classic calorie restriction diet (CR), in which daily caloric intake is normally reduced by 15- 40% [ 10 ], as well as time restriction diet (TR), in which daily food intake is limited to 4–12 h [ 11 ], are among the dietary interventions. Diets that are associated with reduced energy intake exert their positive effects through weight loss and metabolism [ 12 ]. The complete superiority of either regime over the other one has not been proven.…”
Section: Introductionmentioning
confidence: 99%
“…The classic calorie restriction diet (CR), in which daily caloric intake is normally reduced by 15- 40% [ 10 ], as well as time restriction diet (TR), in which daily food intake is limited to 4–12 h [ 11 ], are among the dietary interventions. Diets that are associated with reduced energy intake exert their positive effects through weight loss and metabolism [ 12 ]. The complete superiority of either regime over the other one has not been proven.…”
Section: Introductionmentioning
confidence: 99%
“…Also, previous work by our research team has shown that CR and TR diets can prevent further damage during AKI (47). Possible mechanism(s) of the positive effects of these diets in reducing kidney damage are: improvement of mitochondrial function (19), effect on various transcription factors including FOXO3, HNF4A, HMGA1, and HSF1 (20), increased expression of SIRT1, SIRT3, and activation SOD2 (11,47,48), decreased TGF-β1 and mitochondrial superoxide production and increased GSH concentration (47,49).…”
Section: Discussionmentioning
confidence: 79%
“…Consistent with our results, many studies have shown that different models of dietary restriction are effective in protecting the kidney against AKI: normalization of creatinine levels and reduction of urea and protein excretion in urine after AKI by CR (11,46), protection of the kidney against I/R damage by fasting (19), increased renal resistance to damage by CR and TR (20), and renoprotective effects of IF in diabetes (21). Also, previous work by our research team has shown that CR and TR diets can prevent further damage during AKI (47). Possible mechanism(s) of the positive effects of these diets in reducing kidney damage are: improvement of mitochondrial function (19), effect on various transcription factors including FOXO3, HNF4A, HMGA1, and HSF1 (20), increased expression of SIRT1, SIRT3, and activation SOD2 (11,47,48), decreased TGF-β1 and mitochondrial superoxide production and increased GSH concentration (47,49).…”
Section: Discussionmentioning
confidence: 95%
“…Consistent with our results, many studies have shown that different models of dietary restriction are effective in protecting the kidney against AKI: normalization of creatinine levels and reduction of urea and protein excretion in urine after AKI by ER [ 18 , 66 ], protection of the kidney against I/R damage by fasting [ 26 ], increased renal resistance to damage by ER and TR [ 27 ], and renoprotective effects of IF in diabetes [ 28 ]. Also, previous work by our research team has shown that ER and TR diets can prevent further damage during AKI [ 67 ]. Possible mechanism(s) of the positive effects of these diets in reducing kidney damage are: improvement of mitochondrial function [ 26 ], effect on various transcription factors including FOXO3, HNF4A, HMGA1, and HSF1 [ 27 ], increased expression of SIRT1, SIRT3, and activation SOD2 [ 18 , 67 , 68 ], decreased TGF-β1 and mitochondrial superoxide production and increased GSH concentration [ 67 , 69 ].…”
Section: Discussionmentioning
confidence: 98%
“…Also, previous work by our research team has shown that ER and TR diets can prevent further damage during AKI [ 67 ]. Possible mechanism(s) of the positive effects of these diets in reducing kidney damage are: improvement of mitochondrial function [ 26 ], effect on various transcription factors including FOXO3, HNF4A, HMGA1, and HSF1 [ 27 ], increased expression of SIRT1, SIRT3, and activation SOD2 [ 18 , 67 , 68 ], decreased TGF-β1 and mitochondrial superoxide production and increased GSH concentration [ 67 , 69 ].…”
Section: Discussionmentioning
confidence: 98%