Congenital Tricuspid Insufficiency

Aaron, Benjamin; Mills, Megan; Lower, Richard R.

doi:10.1378/chest.69.5.637

Cited by 24 publications

(19 citation statements)

References 12 publications

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“…10 This case showed a structural abnormality of the prolapsed anterior tricuspid leaflet and absent chordae of the anterior tricuspid leaflet, which is classified as congenital TV dysplasia. 11 Regarding congenital TR, Ebstein anomaly is the most well known of the congenital TV diseases 12 ; however, it is extremely rare, constituting only 0.5% of all congenital heart diseases. 13 TV dysplasia is a congenital structural abnormality of the leaflet, chordae, or papillary muscle associated with the TV and is rarer than Ebstein anomaly.…”

Section: Discussionmentioning

confidence: 99%

Three-Dimensional Echocardiography Reveals Extensive Congenital Anterior Tricuspid Valve Prolapse

Yoshida

Tai

Suzuki-Eguchi

et al. 2020

CASE

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Section: Discussionmentioning

confidence: 99%

Three-Dimensional Echocardiography Reveals Extensive Congenital Anterior Tricuspid Valve Prolapse

Yoshida

Tai

Suzuki-Eguchi

et al. 2020

CASE

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“…An interatrial communication consisting of a pa tent foramen ovale or an ostium secundum atrial septal defect is present in more than half of the cases. About 25% of the patients suffer episodes of paroxysmal atrial tachycardia [2], The electrocardiographic anomalies com monly fall into two categories: those with a right bundle branch block pattern and those with the Wolff-Parkin-White syndrome. The presence of the Wolff-Parkin-White pattern increases the risk of supraventricular paroxys mal tachycardia [3].…”

Section: Discussionmentioning

confidence: 99%

Pregnancy Complicated by Ebstein’s Anomaly: Oxygen Administration to Mother for Chronic Fetal Hypoxemia

Negishi

Yamada²,

Okuyama³

et al. 1995

Fetal Diagn Ther

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For chronic fetal hypoxia due to maternal Ebstein’s anomaly, oxygen was administered daily to the mother by mask for 105 days. At 20 weeks of gestation, umbilical venous blood gases in room air showed pH 7.42, PO₂ 25.7 mm Hg, PCO₂ 33.7 mm Hg and O₂ saturation 48.7%, and changed to 7.45, 39.1 mm Hg, 25.9 mm Hg and 77.4% on 3 liters/min of oxygen inhalation by mask, respectively. The PO₂ of the maternal arterial blood gases increased to 30 mm Hg on oxygen administration at 15 weeks of gestation, but at 25 weeks of gestation the PO₂ increased by only about 10 mm Hg. At 30 weeks, intrauterine growth retardation was suspected. Just after the second puncture of the umbilical cord at 31 weeks and 3 days of gestation, 80 bpm fetal bradycardia occurred for several minutes without recovery and emergency cesarean section was done under the general anesthesia.

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“…On the other hand, the basal attachment of the TV is normal in TV dysplasia. Aaron described the characteristic features of TV dysplasia as follows: [1] focal or diffuse thickening of the leaflets; [2] deficient development of the chordae tendineae and papillary muscles, most often binding down or tethering the valve margin; [3] improper separation of valve components from the ventricular wall; and [4] focal agenesis of valvular tissue [14]. The crucial feature of Ebstein's anomaly is the rotational displacement of the hinge point of the TV leaflet, with maximal apical displacement occurring at the junction of the septal and posterior leaflets and no displacement of the anterior leaflet.…”

Section: Tricuspid Valve Diseasementioning

confidence: 99%

Assessment of Atrioventricular Valve Anatomy and Function in Congenital Heart Diseases Using Three-Dimensional Echocardiography

Nii

2014

Congenital Heart Disease

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The function of the atrioventricular valve (AVV) complex is one of the most important determinants of prognosis in patients with congenital heart disease. However, the anatomy of the AVV complex is complicated, especially in patients with congenital heart disease, which hampers precise preoperative assessment. Moreover, AVV function is maintained by a very delicate balance of the forces generated by the ventricle, atrium, papillary muscles, and blood flow. The AVV leaflet billows with the increment of the hydrostatic pressure in the ventricle and closes by making coaptation with the adjacent leaflets using this balance of force. The shape and size of the annulus or position and function of the papillary muscles are also very important factors in maintaining the effective coaptation of the leaflets with minimal stress on the leaflet and chordae. If a congenital abnormality of the leaflet or valvular apparatus or an incorrect surgical repair causes an imbalance of the forces at work in this delicate system, the result can be valve failure. Since the advent of real-time three-dimensional echocardiography (3DE), we are able to assess the precise anatomical and functional features of this complicated system.

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Congenital Tricuspid Insufficiency

Cited by 24 publications

References 12 publications

Three-Dimensional Echocardiography Reveals Extensive Congenital Anterior Tricuspid Valve Prolapse

Three-Dimensional Echocardiography Reveals Extensive Congenital Anterior Tricuspid Valve Prolapse

Pregnancy Complicated by Ebstein’s Anomaly: Oxygen Administration to Mother for Chronic Fetal Hypoxemia

Assessment of Atrioventricular Valve Anatomy and Function in Congenital Heart Diseases Using Three-Dimensional Echocardiography

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