Corrective treatment of isolated ventricular septal defect in infancy

Agostí, J; Subramanian, S.

doi:10.1016/0022-3468(75)90385-1

Cited by 9 publications

(2 citation statements)

References 17 publications

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“…The occurrence of early postoperative arrthythmias after repair of congenital heart disease was significantly associated with procedure-related risk factors 13 , the presumed mechanism is trauma to the atrioventricular node or the bundle of his before its bifurcation 14,15 . Recent review of primary closure of VSD in infant reported a mortality rate of 2.4% to 24% 6,[9][10][11][12]16,16,17 . The mortality rate in our study was 3.8% which comparable to the published figures.…”

Section: Discussionmentioning

confidence: 99%

Hemodynamic Changes after Surgical Closure of Ventricular Septal Defect.

Haweleh¹

2003

Basrah Journal of Surgery

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This is a prospective study that was carried out during the period between January 1990 to January 2000, to assess the hemodynamic changes and outcome after surgical closure of ventricular septal defect (VSD). Out of 150 patients who underwent surgical closure of VSD, 52 patients had follow up cardiac catheterisation. The main indications for recatheterisation were: persistent respiratory symptoms, clinical or echocardiographic signs of residual leak across the VSD, and improper weight gain. The time interval between recath and surgery was ranging from 3 to 6 months. Complete closure was achieved in 35 patients out of 52 (76%) who had been recatheterized, persistent severe pulmonary hypertension was noticed in one patient who underwent atrial septostomy, 4(8%) patients had significant leak across the defect and high pulmonary hypertension needed a second operation, 12(23%) patients had small residual leak. The mean pulmonary arterial pressure has dropped from 60mmHg to 27 mmHg. Two patients had complete heart block needed an insertion of permanent pacemaker. The hospital mortality of the entire group was 2% and there was no late mortality. In conclusion, hemodynamic changes following the surgical closure of the ventricular septal defect had shown further reduction in the pulmonary pressure and pulmonary vascular resistance as early as 3 months in most patients, rarely some patients needed another attempt of closure of the residual VSD leak. Surgical atrial septostomy might be needed in patients with persistent severe pulmonary hypertension to decompress the right side of the heart.

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Section: Discussionmentioning

confidence: 99%

Hemodynamic Changes after Surgical Closure of Ventricular Septal Defect.

Haweleh¹

2003

Basrah Journal of Surgery

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“…The method of operative intervention at present appears to be less controversial than it was 5 to 10 years ago. Staged treatment with initial pulmonary artery banding as described by Muller and Dammann [1] and later closure debanding and closure of the ventricular septal defect are not as frequently practiced now as they were previously because of the success that has been reported to have been achieved by 1-stage closure of the ventricular septal defect [2,[13][14][15][16][17][18][19][20][21]. The study by Mahle and associates [22] of the 2-stage approach is perhaps the most revealing with regard to the combined mortality of the precedures as well as the included interval mortality while waiting for staged repair.…”

Section: Discussionmentioning

confidence: 99%

Repair of ventricular septal defects

Doty

Lamberth

1985

World j. surg.

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Primary closure of large ventricular septal defects was performed in 57 patients ranging in age from 1 to 24 months during an 11‐year period. Weight of patients ranged from 2.5 to 12.2 kg (mean, 6.6±0.9 kg). Shunt flow was large with mean Qp/Qs, 3.5±0.5. It was possible to close the defect by the transatrial approach in 86% of patients using a prosthetic patch attached to the ventricular septum by continuous polypropylene suture (89%). Early mortality of 5% and late mortality of 2% and excellent functional results in 93% suggest that this approach is appropriate.

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