1996
DOI: 10.1007/bf00352771
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Crystallization behaviour and phase coexistence at morphotrophic phase boundaries in PZT thin films prepared by sol-gel processing

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Cited by 7 publications
(7 citation statements)
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“…However, several groups have produced (100)-oriented films either through seeding or with careful control of the heat-treatment conditions to stabilize an intermediate fluorite or pyrochlore structure that then promotes the more rapid growth of (100)-oriented grains over those with a (111) orientation. [13][14][15][16][17][18][19][20] Films experience many different stresses during processing, resulting from shrinkage and densification, lattice mismatch, and thermal expansion mismatch with the underlying substrate. Garino et al and Tuttle et al were able to quantify these stresses through in situ studies of 20/80 and 53/47 films on platinized Si and MgO.…”
Section: Introductionmentioning
confidence: 99%
“…However, several groups have produced (100)-oriented films either through seeding or with careful control of the heat-treatment conditions to stabilize an intermediate fluorite or pyrochlore structure that then promotes the more rapid growth of (100)-oriented grains over those with a (111) orientation. [13][14][15][16][17][18][19][20] Films experience many different stresses during processing, resulting from shrinkage and densification, lattice mismatch, and thermal expansion mismatch with the underlying substrate. Garino et al and Tuttle et al were able to quantify these stresses through in situ studies of 20/80 and 53/47 films on platinized Si and MgO.…”
Section: Introductionmentioning
confidence: 99%
“…Pb(Zr 53 TI. 47 )TiO 3 thin films with crystallite sizes of 23 nm at 800'C can be prepared by using the methoxyethoxides of the starting elements (20). It was found that the annealing temperature is the important parameter.…”
Section: Sol-gel Processing Of Nanostructured Coatingsmentioning
confidence: 99%
“…In classic CF, lung involvement is the major cause of mortality, dominated by recurrent infections and airway obstruction, finally leading to respiratory failure (Marcorelles et al, 2014). CF is caused by a mutation in the CF transmembrane conductance Regulator gene ( CFTR ; MIM# 602421; GenBank NM 000492.3) which codes for a transmembrane Cl − and HCO 3 − channel (Choi et al, 2001; Ontalus et al, 1996). CFTR gene is located on human chromosome 7, contains 27 exons and generates a 6.4 kb messenger RNA (mRNA; Gregory et al, 1990).…”
Section: Introductionmentioning
confidence: 99%