2019
DOI: 10.1111/bjd.17834
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Cutaneous lymphomas appearing during treatment with biologics: 44 cases from the French Study Group on Cutaneous Lymphomas and French Pharmacovigilance Database

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Cited by 19 publications
(25 citation statements)
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“…Concerning the 27 psoriatic patients receiving biologicals, 18 patients received only 1 biological therapy (etanercept: 6; adalimumab: 5; infliximab: 4; secukinumab: 2; efalizumab: 1) and 9 patients received more than 1 biological therapy (adalimumab: 5; etanercept: 4; infliximab: 2; ustekinumab: 2; apremilast: 2; efalizumab: 1; secukinumab: 1; alefacept: 1; ixekizumab: 1). One additional recent article could not be included in Table due to the lack of specific data …”
Section: Resultsmentioning
confidence: 99%
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“…Concerning the 27 psoriatic patients receiving biologicals, 18 patients received only 1 biological therapy (etanercept: 6; adalimumab: 5; infliximab: 4; secukinumab: 2; efalizumab: 1) and 9 patients received more than 1 biological therapy (adalimumab: 5; etanercept: 4; infliximab: 2; ustekinumab: 2; apremilast: 2; efalizumab: 1; secukinumab: 1; alefacept: 1; ixekizumab: 1). One additional recent article could not be included in Table due to the lack of specific data …”
Section: Resultsmentioning
confidence: 99%
“…The ten above-mentioned cases could be lumped together with the other few reported cases in the literature of psoriasiform MF, a rare variant of MF that simulates psoriasis at the onset. [5][6][7][8][9][10] Finally, it must be underlined that recently, Dequidt et al 69 reported six additional psoriatic patients who developed MF during biological therapies, but due to the lack of specific data about each patient, they could not be included in our review. Interestingly, also Dequidt et al 69 concluded that three scenarios are possible: misdiagnosis of MF as psoriasis; true association MF and psoriasis; and MF de novo, occurring mainly in patients treated for rheumatism or inflammatory colitis.…”
Section: Discussionmentioning
confidence: 99%
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“…[23][24][25] Iatrogenic immunosuppression in early-stage CTCL leads to rapid and dramatic disease exacerbation, underscoring the importance of an intact adaptive immune system for controlling this lymphoma. [26][27][28] Progression of CTCL is associated with an immunosuppressive, inflammatory tumor microenvironment and a loss in T-cell diversity. 22,[29][30][31] Malignant CD4 + T-cells play a role in reorchestrating the immune system from the protective T H 1 response to the T H 2 response characterized by excessive production of inflammatory cytokines including interleukin (IL)-4, IL-5, and IL-10, 32,33 as well as suppression of IL-2 and IFN-γ.…”
mentioning
confidence: 99%