Deep Vein Thrombosis Caused by Congenial Interruption of the Inferior Vena Cava

Tsuji, Yoshihiko; Inoue, Takeshi; Murakami, Hirohisa; Hino, Yutaka; Matsuda, Hitoshi; Okita, Yutaka

doi:10.1177/000331970105201010

Cited by 49 publications

(40 citation statements)

References 17 publications

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“…7 Pyrexia can be present, as described by Tsuji et al, who reported a series of 10 patients where 40% were pyrexic at presentation. 57 …”

Section: Acute Ivc Thrombosismentioning

confidence: 99%

“…Biochemical renal dysfunction may suggest ureteric obstruction secondary to retroperitoneal fibrosis or renal vein thrombosis. 57 Though largely directed from Level IV evidence, it would appear prudent to perform a thrombophilia screen, especially in young patients without other risk factors. This should include: protein C, antithrombin, free protein S, activated protein-C resistance, IgG anticardiolipins, lupus anticoagulant and homocysteine levels as well as a PT (20210) FII gene mutation screen.…”

Section: Haematologicalmentioning

confidence: 99%

“…57 Duplex USS provides an accurate non-invasive method of diagnosing IVC thrombosis and is often the first-line investigative modality. 1 Normality includes a continuous waveform with respiratory variations which become more pulsatile as the IVC empties into the right atrium.…”

Section: Radiologicalmentioning

confidence: 99%

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Inferior vena cava thrombosis: A review of current practice

et al. 2013

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Inferior vena cava (IVC) thrombosis remains under-recognised as it is often not pursued as a primary diagnosis. The aetiology of IVC thrombosis can be divided into congenital versus acquired, with all aetiological factors found among Virchow's triad of stasis, injury and hypercoagulability. Signs and symptoms are related to aetiology and range from no symptoms to cardiovascular collapse. Painful lower limb swelling combined with lower back pain, pyrexia, dilatation of cutaneous abdominal wall veins and a concurrent rise in inflammatory markers are suggestive of IVC thrombosis. Following initial lower limb venous duplex, magnetic resonance imaging (MRI) is the optimal non-invasive imaging tool. Aetiology directs treatment, which ranges from anticoagulation and lower limb compression to open surgery, with endovascular therapies increasingly favoured. The objective of this review is to assess current literature on the aetiology, presentation, investigation, treatment, prognosis and other factors pertaining to IVC thrombosis.

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“…7 Pyrexia can be present, as described by Tsuji et al, who reported a series of 10 patients where 40% were pyrexic at presentation. 57 …”

Section: Acute Ivc Thrombosismentioning

confidence: 99%

Section: Haematologicalmentioning

confidence: 99%

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Inferior vena cava thrombosis: A review of current practice

et al. 2013

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“…14,15 Although less commonly, complete or partial agenesis of the IVC may also present concomitantly with other organ abnormalities, such as congenital heart defects (dextrocardia, septal defects, pulmonary artery stenosis, single atrium, and transposition of great vessels), polysplenia, asplenia, inversion of bowel viscera, and kidney hypoplasia. 15,16 The impact of IVC agenesis in the long-term prognosis of DVT is still unclear. 8 Patients are generally advised to maintain anticoagulation for at least three months following DVT to avoid early recurrence.…”

Section: Discussionmentioning

confidence: 99%

Bilateral Deep Vein Thrombosis Associated with Inferior Vena Cava Agenesis in a Young Patient Manifesting as Low Back Pain

et al. 2017

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Congenital absence of the inferior vena cava is a rare vascular anomaly, and most cases are asymptomatic. Nevertheless, patients with inferior vena cava malformations may have increased risk of deep venous thrombosis. Particularly, cases of bilateral deep venous thrombosis may arise owing to an insufficient collateral venous drainage from the lower limbs. We hereby describe a case of a previously healthy young male patient presenting with bilateral lower limb deep venous thrombosis as the initial clinical manifestation of congenital inferior vena cava agenesis. We conclude that in young patients presenting with deep venous thrombosis, especially when thrombosis occurs spontaneously, bilaterally, or recurrently, inferior vena cava anomalies should be thoroughly investigated and ruled out as appropriate.

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“…4 Recent advances in the utilisation of ultrasound, CT and MRI imaging as well as endovascular procedures have resulted in an increase in detection rates of IVC anomalies as well as the incidental discovery of such abnormalities during unrelated investigations and therapeutic endovascular or surgical procedures. 5 Contrast venography remains the standard for diagnosis of IVC thrombosis with a low false-positive rate and the advantage of access for immediate treatment if required.…”

Section: Investigationsmentioning

confidence: 99%