Demodicidosis Mimicking Granulomatous Rosacea and Transient Acantholytic Dermatosis (Grover’s Disease)

Lindmaier, A; Jurecka, Wolfgang; Lindemayr, H

doi:10.1159/000248826

Cited by 18 publications

(12 citation statements)

References 15 publications

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“…23,24 A GD-like eruption has been linked to demodicidosis. 25 These superficial skin infections likely undermine epidermal integrity inducing GD. When an infection that occurs side by side with GD is cured, epidermal stability is restored and the GD resolves.…”

Section: Discussionmentioning

confidence: 99%

Seasonal variation of transient acantholytic dyskeratosis (Grover's disease)

Scheinfeld¹,

Mones²

2006

Journal of the American Academy of Dermatology

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Section: Discussionmentioning

confidence: 99%

Seasonal variation of transient acantholytic dyskeratosis (Grover's disease)

Scheinfeld¹,

Mones²

2006

Journal of the American Academy of Dermatology

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“…The role of Demodex mites in rosacea and perioral dermatitis remains controversial. A possible etiological role has been suggested in eosinophilic folliculitis [19, 20, 21] and transient acantholytic dermatitis (Grover's disease) [22, 23].…”

Section: Introductionmentioning

confidence: 99%

A clinico‐pathological approach to the classification of human demodicosis

Akilov¹,

Butov²,

Mumcuoglu³

2005

J Deutsche Derma Gesell

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We suggest that demodicosis be divided into both primary and secondary types. The usual etiological agent of primary demodicosis is D. folliculorum, which causes an erythemato-squamous eruption in the facial T-zone. The rash starts on unaltered skin and covers 8 - 15 % of the face. Pruritus accompanies the onset of the rash, while erythema is first apparent after papulo-pustules are seen and disappears after treatment. Half the patients show seasonal exacerbations. Secondary demodicosis is usually caused by D. brevis and characterized by a symmetrical malar papulo-pustular eruption. It develops on diseased skin and covers 30 - 40 % of the face. Pruritus starts after the lesion exacerbation, but erythema precedes the papulo-pustular phase and persists after treatment. Most patients flare during the summer. The facial distribution, seasonality and pathogenesis, as well as the species of mite involved, must be taken into consideration in separating the various forms of demodicosis.

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“…Anhand der bisher recherchierten Fälle können jedoch noch einige spezielle Charakteristika für die hereditäre Form in die Definition aufgenommen werden: Es findet sich zusätzlich häufig eine Hyperhidrosis der Handinnenflächen und Fußsohlen, eine Seborrhö und eine ausgeprägte Pachydermie mit starker Faltenbildung an der Stirn, der sogenannten Cutis verticis et frontis gyrata [10]. Außerdem treten oft Lidverdickungen auf, bis hin zu Gesichtsfeldeinschränkungen [9]. Seltenere Symptome der autosomal dominant vererbten Erkrankung, die ihren Manifestationsgipfel in der Pubertät aufweist, sind bei den wesentlich häufiger betroffenen Männern eine Hypotrichie der Brustbehaarung, eine Gynäkomastie und eine weibliche Verteilung der Schambehaarung [8,9].…”

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“…Außerdem treten oft Lidverdickungen auf, bis hin zu Gesichtsfeldeinschränkungen [9]. Seltenere Symptome der autosomal dominant vererbten Erkrankung, die ihren Manifestationsgipfel in der Pubertät aufweist, sind bei den wesentlich häufiger betroffenen Männern eine Hypotrichie der Brustbehaarung, eine Gynäkomastie und eine weibliche Verteilung der Schambehaarung [8,9]. Die nur selten erkrankten Frauen können zusätzlich unter Regelstörungen leiden [9].…”

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Korrektur der Trommelschlegelfinger unter ästhetischen Aspekten bei hereditärer hypertropher Osteoarthropathie (Touraine-Solente-Gole-Syndrom)

Brüner

Frerichs²,

Raute-Kreinsen³

et al. 2007

Handchir Mikrochir plast Chir

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Finger clubbing can be a single physical finding. In Touraine-Solente-Gole syndrome, the primary form of hypertrophic osteoarthropathy, it is mostly associated with bone pain, hyperhydrosis, pachydermy and wrinkling of the forehead. In other cases, the presence of clubbing is associated with neoplastic, pulmonary, cardiac or other diseases and is then called Bamberger-Pierre-Marie syndrome, the secondary type of hypertrophic osteoarthropathy. The patient's history and careful physical examination, sometimes accompanied by laboratory and imaging studies, leads to the diagnosis. A patient with hereditary hypertrophic osteoarthropathy and its clinical symptoms is presented. Surgical correction of the clubbing fingers is demonstrated in the paper with bilateral resection and shortening of the nail bed, nail matrix and resection of soft tissue. Clubbing fingers are rare, but they might be part of a syndrome or a symptom of other diseases. Reconstructive surgery for aesthetic reasons can be performed.

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Demodicidosis Mimicking Granulomatous Rosacea and Transient Acantholytic Dermatosis (Grover’s Disease)

Cited by 18 publications

References 15 publications

Seasonal variation of transient acantholytic dyskeratosis (Grover's disease)

Seasonal variation of transient acantholytic dyskeratosis (Grover's disease)

A clinico‐pathological approach to the classification of human demodicosis

Korrektur der Trommelschlegelfinger unter ästhetischen Aspekten bei hereditärer hypertropher Osteoarthropathie (Touraine-Solente-Gole-Syndrom)

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