Distemper vaccination in ferrets

Ws, Chalmers; Mr, Geary

doi:10.1136/vr.130.6.127-b

Cited by 3 publications

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“…Several CDV vaccines have been used in domestic ferrets and closely related species in the past. A com- mercial vaccine for mink, 15 experimental vaccinia and canarypox-vectored recombinant vaccines, 1,15 CDV vaccines for dogs, 9,[16][17][18][19] and chick embryo-origin approved ferret vaccines have been used in domestic ferrets. 9 An experimental killed vaccine developed from the Onderstepoort strain of CDV has been used to vaccinate highly susceptible black-footed ferrets (Mustela nigripes) for several years and has been evaluated in Polecat x black-footed ferret hybrids.…”

Section: Discussionmentioning

confidence: 99%

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Wimsatt¹,

Jay²,

Innes³

et al. 2001

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Section: Discussionmentioning

confidence: 99%

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Wimsatt¹,

Jay²,

Innes³

et al. 2001

ajvr

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Dutzler¹

2003

The European System of Central Banks: An Autonomous Actor?

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Subunit‐specific inhibition of inward‐rectifier K⁺ channels by quinidine

et al. 1995

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Distinct inward-rectifier K+ channel subunits were expressed in Xenopus oocytes and tested for their sensitivity to the channel blocker quinidine. The 'strong' inward-rectifier K+ channel IRK1 was inhibited by quinidine with an EC5,, of 0.7 mM, while the 'weak' rectifier channel ROMKl was only moderately inhibited. ROMKI(N171D)-IRKlc_ter, chimeric channels, which carry both sites for strong rectification of IRK1 channels (the negatively charged D171 in the second transmembrane domain and the IRKl-C-terminus including E224), displayed strong rectiticatlon like IRK& but showed weak sensitivity to quinidine-like KOMKl, suggesting independence of quinidine binding and rectitication mechanisms. Moreover, BIRlO and BIRll, two strong rectifier subunits originally cloned from rat brain, exerted subunit-specific sensitivity to quinidine, being much higher for HIRll. Qulnldine blockade of IRK1 was not voltage-dependent, hut strongly dependent on the pH in the superfusate. These results *trongly suggest a subunit-specific interaction of inward-rectifier K+ channels with neutral quinidine within membrane lipid bilaytars.

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Distemper vaccination in ferrets

Cited by 3 publications

References 0 publications

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Checks and Balances

Subunit‐specific inhibition of inward‐rectifier K⁺ channels by quinidine

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Distemper vaccination in ferrets

Cited by 3 publications

References 0 publications

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Serologic evaluation, efficacy, and safety of a commercial modified-live canine distemper vaccine in domestic ferrets

Checks and Balances

Subunit‐specific inhibition of inward‐rectifier K+ channels by quinidine

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About

Subunit‐specific inhibition of inward‐rectifier K⁺ channels by quinidine