Distribution and morphology of enamel hypomineralisation defects in second primary molars

Vlachou, Christina; Arhakis, Aristidis; Kotsanos, Nikolaos

doi:10.1007/s40368-020-00543-9

Cited by 7 publications

(8 citation statements)

References 30 publications

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“…The others are the predictive association between each other and the pattern of dramatically low defect presence in the lingual surfaces of the mandibular teeth, especially so in the anterior teeth. If we consider that HSPM has also shown these similarities to MIH, 6,8‐10 it seems that HSPM, MIH and HOPT belong in the same hypomineralisation spectrum, with causative factors insulting mineralisation of different tooth groups. This has already been suggested for HSPM and MIH 8 and seems that together with HOPT fit into a more general, not yet fully understood, hypomineralisation entity.…”

Section: Discussionmentioning

confidence: 96%

“…HSPM aetiology has been linked to environmental factors without any genetic influence suggested for HSPM so far 7 . Co‐existence and similarity in the clinical and structural appearance between MIH and HSPM, 8‐10 as well as reported similarities in possible aetiological factors, 3 lead to the notion that both belong in the same hypomineralisation spectrum, with aetiological factors for HSPM occurring somewhat earlier in life than for MIH. Most MIH—and many HSPM—studies have examined its presence either in one age group or in different age groups with mixed dentition.…”

Section: Introductionmentioning

confidence: 99%

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Enamel opacities in all other than Molar Incisor Hypomineralisation index teeth of adolescents

Kevrekidou

Kosma

Kotsanos

et al. 2020

Int J Paed Dentistry

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Background There are scarce data on the hypomineralisation of other permanent teeth (HOPT) than the index teeth of Molar Incisor Hypomineralisation (MIH). Aim To report on the prevalence and surface pattern of HOPT and seek associations with MIH. Design Representative samples of urban Greek 14‐year‐olds were examined in classroom with a dental mirror. Their enamel defects were recorded using EAPD criteria for MIH. Descriptive statistics and correlation tests for HOPT vs previously reported MIH findings in the same samples were applied. Results HOPT prevalence in 1156 consented adolescents was 22.9%. 148 (16.2%) of 912 no‐MIH children had HOPT; 117 (48.1%) of the 244 MIH children had HOPT too (OR 3.0, 95% CI 2.4‐3.6). There were more HOPT teeth per child in the MIH vs no‐MIH children (P < .001). HOPT tooth frequency was as follows: second molar 33.7%, canine 25.7%, first premolar 23.6%, and second premolar 17.0%. Enamel breakdown was seen in 5.3% HOPT children. Vestibular to intraoral surface defect ratio differed between jaws (maxilla 187/88, mandible 149/17, P < .05). Conclusions Hypomineralisation defects in the MIH non‐index teeth collectively had comparable prevalence and tooth surface patterns to MIH, but much lower severity. MIH was predictor for HOPT.

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Section: Discussionmentioning

confidence: 96%

Section: Introductionmentioning

confidence: 99%

Enamel opacities in all other than Molar Incisor Hypomineralisation index teeth of adolescents

Kevrekidou

Kosma

Kotsanos

et al. 2020

Int J Paed Dentistry

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“…Vitamin D deficiency due to decreased sun exposure, nutrition problems such as malnutrition and obesity, tobacco and alcohol consumption, complications during pregnancy such as diabetes and hypertension, neonatal complications, low birth weight, and medications during infancy may also have an effect; however, the true causes remain unclear [ 5 , 6 , 7 , 8 ]. Previous studies reported no significant differences in the site of occurrence between the upper and lower regions, between the right and left sides, or between men and women [ 9 ]. Hence, hypomineralization may occur in any person or tooth.…”

Section: Introductionmentioning

confidence: 99%

Evaluation of a Hypersensitivity Inhibitor Containing a Novel Monomer That Induces Remineralization—A Case Series in Pediatric Patients

et al. 2021

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Background: Recently, tooth deformities have been frequently encountered by pediatric dentists. Severe enamel hypomineralization sometimes induces pain such as hyperesthesia, but composite resin restoration is difficult because it often detaches without any cavity preparation. Resin-based hypersensitivity inhibitors for tooth physically seal the dentinal tubules. It was reported that hypersensitivity inhibitor containing novel adhesive monomers forms apatite and induces remineralization in vitro. Therefore, these case series assessed the clinical effects of remineralization and the suppression of hypersensitivity by Bio Coat Ca (Sun Medical, Shiga, Japan). Methods: After mechanical tooth cleaning was performed, the hypersensitivity inhibitors were applied and cured by light exposure. Changes in hypersensitivity were determined by visual analog scale (VAS). The improvement of hypomineralization was evaluated by the change in color tone based on the digital images of intraoral photographs. Results: After repeated monthly treatments, these cases showed decreased hypersensitivity after the fourth application, while the opaque white and brownish color improved on the seventh application. Conclusion: This novel hypersensitivity inhibitor with calcium salt of 4-methacryloxyethyl trimellitic acid (C-MET) and 10-methacryloyloxydecyl dihydrogen calcium phosphate (MDCP) not only suppressed hypersensitivity but also improved cloudiness and brown spots in recently erupted permanent teeth in presented cases.

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“…The term "Molar-Incisor Hypomineralization" (MIH) was presented in 2003, in Athens, being defined as a systemic hypomineralization of one or more permanent first molars with possible involvement of the incisors. In MIH, teeth are characterized by demarcated creamywhite, yellow or brown opacities, post-eruptive enamel breakdown, hypersensitivity, and atypical caries lesion formation [3][4][5][6]. Later, the European Academy of Paediatric Dentistry (EAPD) officially established this condition as MIH [7].…”

Section: Introductionmentioning

confidence: 99%

“…In this sense, the EAPD adopted these diagnostic criteria [1,9,10]. Ever since, multiple studies have studied the etiology of HSPM, pointing to multifactorial, although it is still unclear [2,4,6,8,9,[11][12][13][14][15][16][17][18]. In regard to prevalence, HPSM is significantly reported as low, ranging from 2% to 21.8% [1,5,8,[10][11][12][14][15][16]18,19], though a recent study estimated to be around 4.9% to 9.0% of the worldwide population [6].…”

Section: Introductionmentioning

confidence: 99%

A Four-Year Follow-Up Case Report of Hypomineralized Primary Second Molars Rehabilitated with Stainless Steel Crowns

2021

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Hypomineralized primary second molars (HPSM) are characterized by enamel opacities accompanied by hypersensitivity and atypical caries lesion, on one to four primary second molars. The correct treatment and follow-ups of those teeth have an important impact on a correct eruption of the first permanent molars and future occlusion. Hence, this report aims to describes a case of a severe HPSM in all second molars of a four-year-old girl and subsequent four-year follow-ups. The rehabilitation involved the placement of four stainless steel crowns on all four second primary molars under general anesthesia. Concerning the available literature and the case severity of HPSM, the treatment approach proposed for the case provided good functional outcome.

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Distribution and morphology of enamel hypomineralisation defects in second primary molars

Cited by 7 publications

References 30 publications

Enamel opacities in all other than Molar Incisor Hypomineralisation index teeth of adolescents

Enamel opacities in all other than Molar Incisor Hypomineralisation index teeth of adolescents

Evaluation of a Hypersensitivity Inhibitor Containing a Novel Monomer That Induces Remineralization—A Case Series in Pediatric Patients

A Four-Year Follow-Up Case Report of Hypomineralized Primary Second Molars Rehabilitated with Stainless Steel Crowns

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