Do neighborhood social processes moderate the etiology of youth conduct problems?

Burt, S. Alexandra; Clark, David A.; Pearson, Amber L.; Klump, Kelly L.; Neiderhiser, Jenaè M.

doi:10.1017/s0033291719001521

Cited by 19 publications

(22 citation statements)

References 64 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Instead, its etiology appeared to remain constant regardless of the level of disruption, consistent with research implicating ADHD as a neuropsychiatric diagnosis primarily subject to genetic influences (e.g., 31). These null findings are unlikely to be a function of low statistical power, since prior GxE data simulations ( 32 ) have indicated that researchers have substantial power to detect E moderation in particular. Thus, despite the significant phenotypic association between disruption and scores on hyperactivity/inattention, disruption does not appear to moderate its non-shared environmental etiology.…”

Section: Discussionmentioning

confidence: 93%

Understanding the Effects of the COVID-19 Pandemic on Youth Psychopathology: Genotype–Environment Interplay

Carroll¹,

Shewark²,

Hyde³

et al. 2021

Biological Psychiatry Global Open Science

Self Cite

View full text Add to dashboard Cite

Background Adversity has consistently been found to predict poor mental health outcomes in youth. Perhaps the most omnipresent form of adversity in the last several decades is the coronavirus pandemic of 2020, a global health crisis linked to elevated rates of numerous forms of youth psychopathology. The ongoing nature of the pandemic renders it critical to identify the mechanisms underlying its effects on mental health. Methods The current study examines pandemic-related disruption across multiple domains (e.g., home life, finances) as an etiologic moderator of several common forms of youth psychopathology. Participants were 637 adolescent twin pairs from the Twin Study of Behavioral and Emotional Development in Children (TBED-C). Mothers reported on disruption experienced by the family using the Epidemic-Pandemic Impacts Inventory. Results A series of biometric genotype-environment interaction models revealed that disruption augmented the nonshared environmental contributions to emotional distress and conduct problems, but had little effect on the etiology of attention-deficit hyperactivity problems. Conclusions Our results indicate that identical and fraternal twin similarity in both emotional symptoms and conduct problems decreased with greater disruption, such that children in the same family became less alike, and did so regardless of their degree of genetic resemblance . Put differently, each twin sibling appeared to have their own idiosyncratic experience of pandemic-related disruptions, with downstream consequences for their mental health.

show abstract

Section: Discussionmentioning

confidence: 93%

Understanding the Effects of the COVID-19 Pandemic on Youth Psychopathology: Genotype–Environment Interplay

Carroll¹,

Shewark²,

Hyde³

et al. 2021

Biological Psychiatry Global Open Science

Self Cite

View full text Add to dashboard Cite

show abstract

“…First, this sample is only moderately sized by current twin study standards. However, power analyses (Burt, Clark, Pearson, Klump, & Neiderhiser, 2020; Purcell, 2002) suggest that it may be adequate, particularly since we focused on a reduced AE model. Second, though our enrichment strategy for families from lower‐income neighborhoods was successful, and we used a strong sampling frame from birth records, we did not use sample weights.…”

Section: Discussionmentioning

confidence: 99%

Parenting moderates the etiology of callous‐unemotional traits in middle childhood

Tomlinson

Hyde

Dotterer

et al. 2021

Child Psychology Psychiatry

Self Cite

View full text Add to dashboard Cite

Background Callous‐unemotional (CU) traits are associated with chronic and escalating trajectories of antisocial behavior. Extant etiologic studies suggest that heritability estimates for CU traits vary substantially, while also pointing to an environmental association between parenting and CU traits. Methods We used twin modeling to estimate additive genetic (A), shared environmental (C), and nonshared environmental (E) influences on CU traits, measured with the Inventory of Callous‐Unemotional Traits (ICU) and its subscales. Our sample included 600 twin pairs (age 6–11, 230 monozygotic) from neighborhoods with above‐average levels of family poverty, a risk factor for antisocial behavior. We examined the extent to which correlations between parenting, measured via parent and child report on the Parental Environment Questionnaire, and CU traits reflected genetic versus environmental factors. Then, we tested whether parenting moderated the heritability of CU traits. Results In the context of lower‐income neighborhoods, CU traits were moderately to highly heritable (A = 54%) with similar moderate‐to‐high nonshared environmental influences (E = 46%). Bivariate models revealed that associations between CU traits and warm parenting were genetic (rA = .22) and environmental (rE = .19) in origin, whereas associations between CU traits and harsh parenting were largely genetic in origin (rA = .70). The heritability of CU traits decreased with increasing parental warmth and decreasing harshness. Conclusions Callous‐unemotional traits are both genetic and environmental in origin during middle childhood, but genetic influences are moderated by parenting quality. Parenting may be an important target for interventions, particularly among youth with greater genetic risk.

show abstract

“…Neighborhood disadvantage as measured by the ADI is correlated with poorer physical (Kind et al, 2014; Powell et al, 2020) and mental (Burt et al, 2020) health, as well as higher BMI (Sheets et al, 2020) and lower physical activity (Miller et al, 2021). The ADI also has excellent internal consistency (α = .95 in past research; Singh, 2003).…”

Section: Methodsmentioning

confidence: 99%

Context matters: Neighborhood disadvantage is associated with increased disordered eating and earlier activation of genetic influences in girls.

Mikhail

Carroll

Clark

et al. 2021

Journal of Abnormal Psychology

Self Cite

View full text Add to dashboard Cite

Emerging evidence suggests socioeconomic disadvantage may increase risk for eating disorders (EDs). However, there are very few studies on the association between disadvantage and EDs, and all have focused on individual-level risk factors (e.g., family income). Neighborhood disadvantage (i.e., elevated poverty and reduced resources in one's neighborhood) is associated with increased risk for anxiety/depression and poor physical health. To date, no studies have examined phenotypic associations between neighborhood disadvantage and disordered eating, or how any form of disadvantage may interact with genetic individual differences in risk for EDs. We examined phenotypic and etiologic associations between neighborhood disadvantage and disordered eating in 2,922 girls ages 8-17 from same-sex twin pairs recruited through the Michigan State University Twin Registry. Parents rated the twins on nine items assessing core disordered eating symptoms (e.g., weight preoccupation, binge eating), and neighborhood disadvantage was calculated from 17 indicators of contextual disadvantage (e.g., median home value, neighborhood unemployment). Puberty was measured using the Pubertal Development Scale to examine whether associations were consistent across development. At a phenotypic level, greater neighborhood disadvantage was associated with significantly greater disordered eating symptoms in girls at all stages of puberty (b = .07). Moreover, Genotype 3 Environment models showed that girls living in more disadvantaged neighborhoods exhibited stronger and earlier (i.e., during pre/ early puberty) activation of genetic influences on disordered eating. Results highlight the critical importance of considering contextual disadvantage in research on etiology and risk for disordered eating, and the need for increased screening and treatment for EDs in disadvantaged youth. General Scientific SummaryThis was the first study to examine the association between neighborhood disadvantage and disordered eating in girls. We found that neighborhood disadvantage was associated with significantly greater disordered eating symptoms across all stages of puberty in girls, and that girls living in disadvantaged neighborhoods exhibited stronger and earlier (i.e., during pre/early puberty) activation of genetic influences on disordered eating. Results indicate that neighborhood disadvantage is a critical contextual factor with implications for etiology and risk for disordered eating in girls.

show abstract

Do neighborhood social processes moderate the etiology of youth conduct problems?

Cited by 19 publications

References 64 publications

Understanding the Effects of the COVID-19 Pandemic on Youth Psychopathology: Genotype–Environment Interplay

Understanding the Effects of the COVID-19 Pandemic on Youth Psychopathology: Genotype–Environment Interplay

Parenting moderates the etiology of callous‐unemotional traits in middle childhood

Context matters: Neighborhood disadvantage is associated with increased disordered eating and earlier activation of genetic influences in girls.

Contact Info

Product

Resources

About