Drug-induced linear IgA bullous dermatosis probably induced by furosemide

Cerottini, Jean‐Philippe; Ricci, C; Guggisberg, D.; Panizzon, Renato G.

doi:10.1016/s0190-9622(99)70414-7

Cited by 39 publications

(19 citation statements)

References 15 publications

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“…Indirect IF on salt‐split skin reveals an epidermal binding pattern in the majority of cases, while a few cases show combined epidermal and dermal binding, and other cases a purely dermal binding pattern. Indirect IF has sometimes failed to identify circulating antibodies in drug‐induced LABD (11, 12, 17, 26–28, 36, 39–40). These findings suggest that the target antigens of drug‐induced LABD are heterogenous.…”

Section: Discussionmentioning

confidence: 99%

Drug-Induced Linear IgA Bullous Dermatosis

Onodera

Mihm

Yoshida

et al. 2005

The Journal of Dermatology

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We report the case of a 69-year-old Japanese woman with multiple blistering lesions covering almost her whole body. Linear IgA and C3 depositions were seen at the basement membrane zone on direct immunofluorescence (IF). Linear IgA bullous dermatosis (LABD) is one of the autoimmune diseases resulting in subepidermal blisters. It is clinically similar to bullous pemphigoid and IF is required to distinguish the two diseases. In this case, the blistering lesions appeared after vancomycin treatment. This drug was strongly suspected as a cause of LABD in light of the clinical course of the patient even though a drug-lymphocyte stimulating test was negative. Among the various implicated causative drugs, vancomycin is the most commonly associated with LABD.

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Section: Discussionmentioning

confidence: 99%

Drug-Induced Linear IgA Bullous Dermatosis

Onodera

Mihm

Yoshida

et al. 2005

The Journal of Dermatology

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“…Although LABD is usually idiopathic, it may be induced by drugs, malignancies, or infections. Many drugs, such as acetaminophen 2 , amiodarone 3 , furosemide 4 , and phenytoin, 5 have been implicated as the cause; however, vancomycin has been found to be the one most commonly associated 6 . To our knowledge, there have been two cases of vancomycin-induced LABD reported in Korea to date.…”

Section: Introductionmentioning

confidence: 99%

Vancomycin-induced Linear IgA Bullous Dermatosis: A Case Report and Review of the Literature

2008

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Linear IgA bullous dermatosis (LABD) is a rare autoimmune bullous disease that can either occur without any apparent cause or be induced by the administration of certain drugs, the most common of which is vancomycin. We present a case of a 45-year-old woman who was diagnosed with vancomycin-induced LABD by the presence of a characteristic linear band of IgA along the basement membrane zone on direct immunofluorescence microscopy. Our patient showed complete recovery after a 2-week period during which vancomycin administration was discontinued. (Ann Dermatol (Seoul) 20(2) 102∼106, 2008)

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“…Linear IgA bullous dermatosis (LABD) is an autoimmune bullous disease, most commonly involving the trunk, the proximal areas of the limbs and the acral regions . LABD is characterized by the presence of subepidermal bullae and by homogenous linear IgA deposition along the basement membrane zone . LABD is often idiopathic, but some cases may be drug‐induced .…”

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confidence: 99%

“…LABD is often idiopathic, but some cases may be drug‐induced . Some of the suspected drugs are vancomycin, interferons, furosemide and piroxicam . We report a patient with moxifloxacin‐induced LABD.…”

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confidence: 99%

“…Vancomycin is the most comon drug associated with LABD. Other associated drugs are penicillin, ceftriaxone, metronidazole, piroxicam, diclofenac, vigabatrin, phenytoin, rifampin, amiodarone, furosemide, lithium carbonate, somatostatin, interferon‐α 2a and atorvastatin . Moxifloxacin is a third‐generation fluroquinolone antibiotic with Gram‐positive activity, which is commonly used for treatment of respiratory tract infections.…”

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confidence: 99%

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