Effect of prostaglandin A1 on renal hemodynamics in pregnancy toxemia

Toppozada, M.; Ghoneim, A.; Habib, Y. A.; El-Ziadi, L.; El-Damarawy, H.

doi:10.1016/s0002-9378(16)32980-5

Cited by 17 publications

(2 citation statements)

References 12 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Changes in renal function have been implicated in the development of hypertension. The hypertensive effect may be me diated by severe alterations in renal prostaglandin syn thesis [16]. Such an imbalance in the production of pros tacyclin and thromboxane, leading to increased vasocon striction.…”

Section: Discussionmentioning

confidence: 99%

The Association of Pregnancy-Induced Hypertension and Uterine Malformations

Ben‐Rafael

Seidman

Recabi

et al. 1990

Gynecol Obstet Invest

View full text Add to dashboard Cite

To evaluate the incidence of pregnancy-induced hypertension (PIH) in women with congenital uterine malformations, we examined the pregnancy complications of 67 women with uterine anomalies demonstrated by hysterosalpingography (HSG). The study group was compared with a control group of 130 women with normal-shaped uterus proven by HSG – matched for age, parity and presenting complaint. A significantly increased (p < 0.04) rate of PIH was found for women with uterine malformation as well as a 2-fold higher frequency of preeclampsia. The etiology for this association is unknown, however; it is suggested that uterine malformation is a predisposing condition to PIH.

show abstract

Section: Discussionmentioning

confidence: 99%

The Association of Pregnancy-Induced Hypertension and Uterine Malformations

Ben‐Rafael

Seidman

Recabi

et al. 1990

Gynecol Obstet Invest

View full text Add to dashboard Cite

show abstract

“…Injections of pharmacologic doses of PG [19][20][21] or substances that block synthesis of Tx [22][23][24] have been shown to have beneficial effects both in various models of renal disease and in humans with diminished renal function due to preeclampsia [25] and chronic glomeru lonephritis [26]. PUFAs may act by a similar mechanism, since y-linolenic, linoleic, and eicosapentaenoic acids are precur sors of the mono- [27], di- [28], and trienoic [29,30] Ps, respectively.…”

mentioning

confidence: 99%