Effect of vitamin E on oxidative stress status in small intestine of diabetic rat

Abstract

The results revealed the occurrence of oxidative stress in the small intestine of diabetic rats. Vitamin E, as an antioxidant, attenuates lipid peroxidation and protein oxidation, and increases antioxidant defense mechanism.

Regenerative Potential of D- and #948;-Tocotrienol Rich Fraction on Crushed Skeletal Muscle of Diabetic Rats

“…Elsy, et al: Vitamin E and muscle regeneration in diabetics lower in DC, this is in agreement with other studies [30,31]. This activity was normalizing in control group after vitamin E treatment [31].…”

“…This activity was normalizing in control group after vitamin E treatment [31]. The 3-week administration of d-δ-TRF enhances the serum catalase activity in treated groups [6].…”

Therapeutic potential of d-δ-tocotrienol rich fraction on excisional skin wounds in diabetic rats

“…The decreased catalase activity in plasma and tissues of STZ-diabetic rats may be due to its increased utilization for scavenging the toxic products of lipid peroxidation or due to decreased availability of H 2 O 2 [49]. Vitamin E treatment has been shown to normal ize the catalase activity in the control group [50]. The result of present study revealed that d-δ-TRF supplementation enhances the serum catalase activity in treated groups.…”

“…The present study showed that the serum catalase activity was lower in DC. Many other studies [49,50] stated that the catalase activity had decreased in plasma, liver and kidney of diabetic control rats. The decreased catalase activity in plasma and tissues of STZ-diabetic rats may be due to its increased utilization for scavenging the toxic products of lipid peroxidation or due to decreased availability of H 2 O 2 [49].…”

Effects of Quercetin-Supplementation in NADH-Diaphorase Positive Neurons Subpopulations in the Ileum of Rats with Experimental Diabetes Mellitus

“…The combination of intracellular signaling disorders (De Giorgio & Camilleri, 2004), morphoquantitative and neurochemical changes of enteric neurons may be linked to relevant clinical problems of neurological manifestations of DM. Therapeutic measures are being concentrated in an attempt to prevent and/or reduce the excess of free radicals generated during the oxidative stress, in order to prevent or lessen the neurological diabetes complications (Shirpoor et al, 2007).…”