Embolization of Ruptured Hepatic Hydatid Cyst to Pulmonary Artery in an Elderly Patient : Multidetector Computed Tomography Findings = عملية تسديد تكيس عداري كبدي متمزق إلى الشريان الرئوي في مريض مسن

Özkan, Fuat; Yesilkaya, Yakup; Tokur, Mahmut; Özcan, Nuri; İnci, Mehmet Fatih

doi:10.12816/0003214

Cited by 2 publications

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“…The dog is then contaminated by eating the discarded offal of sheep. Humans are an accidental intermediate host that is contaminated by the ingestion of embryophores released in the excrement of parasitized dogs, either by direct contact or by ingestion of contaminated water or raw food 8 …”

Section: Discussionmentioning

confidence: 99%

Four pathways for hydatid pulmonary embolism

et al. 2020

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Introduction Hydatid pulmonary embolism (HPE) is rare but remains an etiology that needs to be considered and kept in mind, especially in endemic areas, as regards nonthrombotic embolism. Methods Between 2000 and 2014, 10 patients were treated in our department for the presence of hydatid material in the pulmonary arterial circulation. Through these 10 cases, we try to assess the radioclinical characteristics and results of the surgical treatment. Results The study included six men and four women, average age 28 years. Discovery of HPE was made following the exploration of cardiorespiratory symptoms in nine cases and was incidental in a single patient. Computed tomography angiography (CTA) of the chest directly visualized the hydatid material in the pulmonary arteries in nine cases. Surgery consisted of an embolectomy under cardiopulmonary bypass in nine cases. This embolectomy was preceded by the treatment of an embolic hydatid cyst (HC) in seven cases (HCs of the right heart chambers in six cases and a liver HC ruptured in the inferior vena cava in one case), whereas for only one patient, who presented a HC pedunculated in the right ventricle and protruding into the pulmonary artery, the cure of the cardiac location was sufficient. Only one patient died on the third postoperative day. Conclusion Endoluminal hydatid involvement of pulmonary arteries is extremely rare. CTA chest has an important contribution to the positive diagnosis. Treatment is surgical when the patient's condition allows it.

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Section: Discussionmentioning

confidence: 99%

Four pathways for hydatid pulmonary embolism

et al. 2020

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“…which are: pulmonary embolism [12,13], acute pericarditis [8,23,24], endocavitary rupture with anaphylactic shock, massive embolism or blockage of a valvular orifice [14]. On the electrocardiogram, signs of myocardial necrosis may be due to thinning of the myocardial layers surrounding the cyst, or secondary to obstruction of the coronary vessels by daughter vesicles [11]; a true necrotic Q wave can also be seen [11,23].…”

Section: Observationmentioning

confidence: 99%

“…Cardiac hydatidosis is a condition characterized by its long functional tolerance and its clinical and para-clinical polymorphism, thus representing a real diagnostic and therapeutic challenge that is sometimes difficult to resolve [3][4][5][6][7][8].It is particularly serious and severe because of its possible complications: acute pericarditis [8], tamponade [9], arrhythmia [10], infarction [11], pulmonary embolism [12,13], cardiac arrest , and sudden death [14].…”

Section: Introductionmentioning

confidence: 99%

An Unexpected Etiology of Myocardial Ischemia in Young

Bouomrani

Souissi

Guermazi

et al. 2019

JHC

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Introduction The cardiac localization of hydatid cyst (HC) is rare and little known. It is exceptionally primary and isolated. Myocardial ischemia remains an exceptional and unusual manifestation of this localization. We report the original observation of cardiac HC revealed by acute myocardial ischemia in a young subject. Observation A 35-year-old patient with no notable pathological history was admitted to our department via the emergency room for acute anterior chest pain. The somatic examination was without abnormalities as well as the baseline biological tests. The electrocardiogram demonstrated epicardial ischemia in the infero-lateral territory without other abnormalities. The cardiac troponin I was slightly elevated (32ng/l, N<19ng/l). Subsequent controls after 30min, 60min and 180min did not show significant variations. Subsequent investigations (CT, coro-CT, coronary angiography) led to the diagnosis of intramyocardial HC of the left ventricle, with no abnormalities of the coronary arteries. After surgical excision, the evolution was favorable with normalization of the electrocardiogram and the troponin Ic. Conclusion Cardiac localization of this parasitosis is exceptional and characterized by an important clinical polymorphism making its diagnosis a real challenge. It seems useful to discuss the diagnosis of cardiac HC in front of any unexplained electrocardiogram abnormality occurring in endemic zone.

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Embolization of Ruptured Hepatic Hydatid Cyst to Pulmonary Artery in an Elderly Patient : Multidetector Computed Tomography Findings = عملية تسديد تكيس عداري كبدي متمزق إلى الشريان الرئوي في مريض مسن

Cited by 2 publications

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Four pathways for hydatid pulmonary embolism

Four pathways for hydatid pulmonary embolism

An Unexpected Etiology of Myocardial Ischemia in Young

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