Endothelial Dysfunction in Passive Smokers

Leone, Aurelio

doi:10.15406/jccr.2014.01.00039

Cited by 9 publications

(6 citation statements)

References 43 publications

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“…Similar results were reported by the studies of Celermajer et al [19], and Giannini et al [20]. In addition a very recent review of Leone [21] gave effectiveness to these results, also discussing the role of nicotine-enantiomers. It would seem a logical assumption that a reinforced power of the Nicotine-isomer (+), having a wellknown unpleasant taste in the smoked cigarette, could reduce nicotine desire and, consequently, the smoking habit.…”

Section: Review Articlesupporting

confidence: 83%

Smoking and Hypertension

2015

JCCR

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Smoking and hypertension are two well-known independent risk factors for both heart and blood vessel. A large number of observations identify cigarette smoke as a factor able to cause a functional and initially transient damage primarily of the endothelium and reduced tolerance to exercise stress testing because of the effects of nicotine and carbon monoxide. At the time, the functional damage became an irreversible pathological damage with ischemic lesions of the myocardium and artery vessel atherosclerosis. In its turn, hypertension plays harmful effects on the heart, kidney and arterial tree, mainly coronary, carotid and cerebral vascular structures, by its complications, the target organs of which are the same of cigarette smoke. There is evidence that the association of cigarette smoking with hypertension exponentially increases the risk of cardiovascular disease and events when compared to that of each of these factors singly acting.

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Section: Review Articlesupporting

confidence: 83%

Smoking and Hypertension

2015

JCCR

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“…The primary pathological mechanism to explain these responses is related to the role of adrenergic and sympathetic system, which play a different activity on BP in the morning and night. Thus, it is worth noting that S strongly feels the effects of the last two parameters, which are influenced by nicotine and carbon monoxide [9]. Therefore, the adverse relationship between S and BP cannot be absolutely denied as this finding undoubtedly shows…”

Section: Resultsmentioning

confidence: 93%

Evidence of Impaired Night-Response of Blood Pressure in Hypertensive Smokers. An Update*

Leone¹

2015

JCCR

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Submit Manuscript | http://medcraveonline.com J Cardiol Curr Res, 4(2): 00138 Abbreviations: S: Smoking; H: Hypertension; BP: Blood Pressure; ABPM: Ambulatory Blood Pressure Monitoring ABPM 104 (100%) Mean systolic BP (mmHg, smokers) 162+/-18 Mean diastolic BP (mmHg, smokers) 92 +/-7 Mean systolic BP (mmHg, non-smokers) 159+/-17 Mean diastolic BP (mmHg, non-smokers) 94 +/-6 Research Article AbstractEpidemiological survey has demonstrated an impaired night response of BP in hypertensive smokers.This Ambulatory Blood Pressure Monitoring Study (ABPM) conducted in hypertensive chronic smokers and updated by the analysis of more recent findings undoubtedly shows that a large majority of these individuals belongs to the group of non-dipper with a nocturnal fall in BP more than 10%.

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“…It has been recognized that a strong interaction exists between the major cardiovascular risk factors [1][2][3], which may exert their harmful effects on cardiovascular system either independently or variably associated with statistical, probabilistic evidence, in this case, an exponential increased rate of disease and, consequently, more powerful response. A different matter concerns the effects of lack or changing a cardiovascular risk factor, being this occurrence not yet well established.…”

Section: Editorialmentioning

confidence: 99%

Endothelial Dysfunction in Passive Smokers

Cited by 9 publications

References 43 publications

Smoking and Hypertension

Smoking and Hypertension

Evidence of Impaired Night-Response of Blood Pressure in Hypertensive Smokers. An Update*

How does to Quit Smoking Influence the Effects of Other Major Cardiovascular Risk Factors?

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