Epidermodysplasia verruciformis mimicking pityriasis versicolor

Zhang, Bin; Huan, Xing; He, Rui; Li, Song; Ma, Lin

doi:10.1002/ped4.12288

Cited by 1 publication

(1 citation statement)

References 5 publications

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“…[117][118][119][120][121][122][123][124] On the other hand, hyperpigmented lesions of tinea versicolor should be differentiated from tinea corporis, pityriasis rosea, pityriasis rotunda, tinea imbricata, acanthosis nigricans, terra firma-forme dermatosis, café au lait macules, ephelides, solar lentigines, melasma, erythrasma, guttate psoriasis, nummular eczema, seborrheic dermatitis, contact dermatitis, post-inflammatory hyperpigmentation, type 1 (classic adult) pityriasis rubra pilaris, secondary syphilis, confluent and reticulated papillomatosis (also known as Gougerot-Carteaud syndrome), and epidermodysplasia verruciformis (also known as Lewandowsky-Lutz dysplasia). [125][126][127][128][129][130][131][132][133][134][135][136][137][138] The distinctive features of many of these conditions help to differentiate them from tinea versicolor.…”

Section: Differential Diagnosismentioning

confidence: 99%

Tinea versicolor: an updated review

Leung¹,

Barankin²,

Lam³

et al. 2022

DIC

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A search was conducted in July 2022 in PubMed Clinical Queries using the key terms "tinea versicolor" OR "pityriasis versicolor". The search strategy included all clinical trials (including open trials, non-randomized controlled trials and randomized controlled trials), observational studies and reviews (including narrative reviews and meta-analyses) published within the past 10 years. Only papers published in the English literature were included in this review. The information retrieved from the search was used in the compilation of this article. Review AetiopathogenesisTinea versicolor is caused by dimorphic lipophilic and lipid-dependent yeasts in the genus Malassezia (formerly known as Pityrosporum) species, notably Malassezia globosa (M. globose), M. furfur and M. sympodialis. [2][3][4][5][6][7][8][9][10][11][12] Other species that have been implicated include M. restricta, M. obtuse, M. slooffiae, M. pachydermatis and M. japonica. [13][14][15][16] These yeasts are normal commensals on the skin surface. 17,18 Skin colonization increases with age; 25% of children and almost 100% of adults are affected. 19 Tinea versicolor occurs when the saprophytic yeast or budding form of the organism converts to the pathogenic hyphal or mycelial form. The fungal infection is localized to the stratum corneum. Predisposing factors for the conversion include a hot and humid environment, hyperhidrosis, application of oily lotion or cream to the skin, wearing of masks, excessive lipid-containing sebaceous secretions, malnutrition, poor general health, use of oral contraceptives, pregnancy, diabetes mellitus, use of topical or systemic corticosteroids, Cushing disease, Helicobacter pylori infection, immunodeficiency and genetic predisposition. [20][21][22][23][24][25][26][27][28][29][30][31][32][33][34][35][36] A recent study showed oxidative stress has no role in the pathogenesis of tinea versicolor. 29

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