Erratum: Dual effect of erythropoietin on liver protection and regeneration after subtotal hepatectomy in rats

Greif, Franklin; Ben‐Ari, Ziv; Taya, Rasim; Pappo, Orit; Kurtzwald, Efrat; Cheporko, Yelena; Ravid, Amiram; Hochhauser, Edith

doi:10.1002/lt.22100

Liver Transpl

2010

DOI: 10.1002/lt.22100

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Erratum: Dual effect of erythropoietin on liver protection and regeneration after subtotal hepatectomy in rats

Franklin Greif¹,

Ziv Ben‐Ari²,

Rasim Taya³

et al.

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Cited by 7 publications

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Acute-on-chronic Liver Failure

Sarin

Choudhury

2016

Curr Gastroenterol Rep

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Acute-on-chronic liver failure (ACLF) is a distinct entity that differs from acute liver failure and decompensated cirrhosis in timing, presence of treatable acute precipitant, and course of disease, with a potential for self-recovery. The core concept is acute deterioration of existing liver function in a patient of chronic liver disease with or without cirrhosis in response to an acute insult. The insult should be a hepatic one and presentation in the form of liver failure (jaundice, encephalopathy, coagulopathy, ascites) with or without extrahepatic organ failure in a defined time frame. ACLF is characterized by a state of deregulated inflammation. Initial cytokine burst presenting as SIRS, progression to CARS and associated immunoparalysis leads to sepsis and multi-organ failure. Early identification of the acute insult and mitigation of the same, use of nucleoside analogue in HBV-ACLF, steroid in severe alcoholic hepatitis, steroid in severe autoimmune hepatitis and/or bridging therapy lead to recovery, with a 90-day transplant-free survival rate of up to 50 %. First-week presentation is crucial concerning SIRS/sepsis, development, multiorgan failure and consideration of transplant. A protocol-based multi-disciplinary approach including critical care hepatology, early liver transplant before multi-organ involvement, or priority for organ allocation may improve the outcome. Presentation with extrahepatic organ involvement or inclusion of sepsis as an acute insult in definition restricts the therapy, i.e., liver transplant or bridging therapy, and needs serious consideration. Augmentation of regeneration, cell-based therapy, immunotherapy, and gut microbiota modulation are the emerging areas and need further research.

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Acute-on-chronic Liver Failure

Sarin

Choudhury

2016

Curr Gastroenterol Rep

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Glycoengineering tobacco plants to stably express recombinant human erythropoietin with different N-glycan profiles

Kittur

Hung

Zhu

et al. 2020

International Journal of Biological Macromolecules

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Effect of erythropoietin on liver regeneration in an experimental model of partial hepatectomy

Gül

Comert

Çakmak

et al. 2013

International Journal of Surgery

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Erratum: Dual effect of erythropoietin on liver protection and regeneration after subtotal hepatectomy in rats

Cited by 7 publications

References 0 publications

Acute-on-chronic Liver Failure

Acute-on-chronic Liver Failure

Glycoengineering tobacco plants to stably express recombinant human erythropoietin with different N-glycan profiles

Effect of erythropoietin on liver regeneration in an experimental model of partial hepatectomy

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