Erratum: Metabolic effects of smoking cessation

Harris, Kindred K.; Zopey, Mohan; Friedman, Theodore C.

doi:10.1038/nrendo.2016.171

Cited by 21 publications

(20 citation statements)

References 1 publication

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“…Recent evidence indicates that the risk of developing type 2 diabetes is 30–40% higher in smokers that nonsmokers [ 9 ]. Passive (second-hand) smoking has also been linked with negative health consequences such as low-birth rate in offspring of mothers exposed to second-hand smoke, sudden infant death syndrome, and type 2 diabetes mellitus [ 28 ].…”

Section: Health Effects Of Cigarette (Tobacco) Smokingmentioning

confidence: 99%

Clinical Effects of Cigarette Smoking: Epidemiologic Impact and Review of Pharmacotherapy Options

Onor

Stirling

Williams

et al. 2017

IJERPH

126

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Cigarette smoking—a crucial modifiable risk factor for organ system diseases and cancer—remains prevalent in the United States and globally. In this literature review, we aim to summarize the epidemiology of cigarette smoking and tobacco use in the United States, pharmacology of nicotine—the active constituent of tobacco, and health consequence of cigarette smoking. This article also reviews behavioral and pharmacologic interventions for cigarette smokers and provides cost estimates for approved pharmacologic interventions in the United States. A literature search was conducted on Google Scholar, EBSCOhost, ClinicalKey, and PubMed databases using the following headings in combination or separately: cigarette smoking, tobacco smoking, epidemiology in the United States, health consequences of cigarette smoking, pharmacologic therapy for cigarette smoking, and non-pharmacologic therapy for cigarette smoking. This review found that efficacious non-pharmacologic interventions and pharmacologic therapy are available for cessation of cigarette smoking. Given the availability of efficacious interventions for cigarette smoking cessation, concerted efforts should be made by healthcare providers and public health professionals to promote smoking cessation as a valuable approach for reducing non-smokers’ exposure to environmental tobacco smoke.

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Section: Health Effects Of Cigarette (Tobacco) Smokingmentioning

confidence: 99%

Clinical Effects of Cigarette Smoking: Epidemiologic Impact and Review of Pharmacotherapy Options

Onor

Stirling

Williams

et al. 2017

IJERPH

126

View full text Add to dashboard Cite

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“…29) Various toxic substances, such as nicotine, carbon monoxide and oxidizing gas from cigarette, cause abnormalities in lipoprotein metabolism, endothelial cell dysfunction and insulin resistance, which increase the risk of MetS. 30) 31) 32) 33) 34) Nicotine stimulates the release of adrenaline, which increases blood pressure and heart rate. 35) It has been suggested that nicotine induces lipolysis in adipose tissue, increases triglyceride, and low-density lipoprotein (LDL) cholesterol, and decrease HDL cholesterol in the blood.…”

Section: Smoking Cessationmentioning

confidence: 99%

“… 34) The risk of MetS due to smoking increases in proportion to the amount of smoking. 33) Smoking cessation reduces the risk of developing MetS, cardiovascular disease and mortality, 31) 38) 39) so smoking cessation should be emphasized. However, for some time after quitting smoking, weight gain may occur, so lifestyle modification should be implemented more thoroughly after smoking cessation.…”

Section: Smoking Cessationmentioning

confidence: 99%

“…lowers stress, suppresses the desire to smoke and reduces withdrawal symptoms; therefore, it is effective in maintaining smoking cessation. 31) 47) 48) Since weight gain after smoking cessation usually occurs, 31) 49) it is important to maintain appropriate weight through regular exercise and healthy eating habits. 31) 47) 48) Avoiding fatty, sweet and high-calorie foods as well as making a habit of eating vegetables and fruits every day is helpful in smoking cessation.…”

Section: Smoking Cessationmentioning

confidence: 99%

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Lifestyle Modification in the Management of Metabolic Syndrome: Statement From Korean Society of CardioMetabolic Syndrome (KSCMS)

et al. 2022

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Author's summary Metabolic syndrome (MetS) is highly prevalent and is associated with worse cardiovascular outcome. Lifestyle modification is the most effective way to reduce the incidence of cardiovascular complications caused by MetS. Here, we would like to review the effects and specific methods of the 6 lifestyle modifications (weight control, smoking cessation, alcohol drinking in moderation, diet control, exercise and physical activity, and cognitive behavioral therapy) and control of blood pressure, dyslipidemia, and blood sugar based on evidence in the management of MetS.

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“…Smoking cessation alleviates diseases caused by smoking (1,2), and the reduction of oxidative damage is one expected benefit (3). Past intervention studies reported that smoking cessation yielded reduced levels of urinary 8-hydroxy-2′-deoxyguanosine (8-OHdG) and 8-iso-prostaglandin F2α (8-isoprostane) (3,4).…”

Section: Introductionmentioning

confidence: 99%

Changes in oxidative stress levels during two weeks of smoking cessation treatment and their association with nutritional characteristics in Japanese smokers

et al. 2019

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Although several experimental studies have reported that oxidative stress levels decrease during smoking cessation, how they change among general smokers has yet to be completely elucidated. In the present study, a total of 23 smokers who underwent smoking cessation treatment were observed for two-week changes in their levels of 8-OHdG and 8-isoprostane. Physical and nutritional characteristics were measured at the initial patient visit, and casual urine samples were collected at the initial visit and at a follow-up visit two weeks later. Oxidative stress was measured by a high performance liquid chromatography electrochemical detector, and the two-week difference in the levels of oxidative stress was assessed according to demographic and nutrient factors. Neither the urinary level of 8-OHdG nor that of 8-isoprostane decreased, although the cotinine level was decreased at two weeks. A Two-way repeated ANOVA revealed a significant interaction for fat intake by time for the change in the 8-OHdG level (P=0.03) and significant interactions for α-tocopherol intake (P=0.03), iron intake, and carbohydrate intake (P=0.03), all of which were time-dependent for the change in the 8-isoprostane level. The 8-OHdG level decreased among smokers with a high fat intake and was increased with a low fat intake. The 8-isoprostane levels were decreased among smokers with a high carbohydrate intake and increased with a low carbohydrate intake, decreased with a low iron intake and increased with a high iron intake and decreased with a low α-tocopherol intake and increased with a high α-tocopherol intake. Although the present study failed to observe a decrease in oxidative stress levels during the two-week smoking cessation period, we hypothesize that the intake levels of specific nutrients when initiating smoking cessation treatment may predict any subsequent changes in the oxidative stress levels.

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Erratum: Metabolic effects of smoking cessation

Cited by 21 publications

References 1 publication

Clinical Effects of Cigarette Smoking: Epidemiologic Impact and Review of Pharmacotherapy Options

Clinical Effects of Cigarette Smoking: Epidemiologic Impact and Review of Pharmacotherapy Options

Lifestyle Modification in the Management of Metabolic Syndrome: Statement From Korean Society of CardioMetabolic Syndrome (KSCMS)

Changes in oxidative stress levels during two weeks of smoking cessation treatment and their association with nutritional characteristics in Japanese smokers

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