Ethylene glycol poisoning

Parry, Michael F.; Wallach, Ronald

doi:10.1016/0002-9343(74)90780-3

Cited by 214 publications

(60 citation statements)

References 32 publications

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“…In the clinical course this case showed different features from other cases (1-3). The symptomsof ethylene glycol poisoning consist of three stages in the clinical course: In the first stage central nervous system (CNS) symptoms appear, in the second stage cardiopulmonary dysfunction becomesthe main problem and in the third stage renal dysfunction develops (2). In the present case directly-absorbed oxalate caused acute renal failure without severe CNSand cardiopulmonary symptoms.…”

Section: Discussionmentioning

confidence: 63%

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Acute Renal Failure due to Oxalate Ingestion.

et al. 1998

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A 47-year-old man presented with acute renal failure following oxalate ingestion. Nausea and hematoemesis appeared four hours after attempted suicide and acute oliguric renal failure ensued the following day. The patient underwent four sessions ofhemodialysis and then reverted to normal state. Histopathologic examination of renal biopsy specimen revealed the degeneration of the renal tubular epithelial cells associated with intracellular calcium oxalate crystal deposition. Most of the renal tubules were patent despite the intraluminar crystal deposition. These findings suggest that dysfunction of the renal tubular epithelial cell plays a more important role than tubular obstruction in developing acute renal failure. (Internal Medicine 37: 762-765, 1998)

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Section: Discussionmentioning

confidence: 63%

“…Renal failure due to intratubular oxalate crystals has been reported in cases after massive ascorbic acid administration ( 1 ) and ethylene glycol intoxication (2). Wetreated a case with acute renal failure caused by ingestion of oxalate, which however has not ever been documented.…”

Section: Introductionmentioning

confidence: 89%

Acute Renal Failure due to Oxalate Ingestion.

et al. 1998

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“…The signs as noticed here have been reported by Crowell et al (1979) in cattle and by Paton (1989) and Herd (1992) in small animals. Anuria may be due t o mechanical blockage of renal tubules by the deposition of calcium oxalate crystals (Moriarty and Mc Donald, 1974;Parry and Wallach, 1974) which are formed by combination of oxalate (end product of ethylene glycol) and calcium ions present in circulation (Oehme, 1974). Occurrence of anuria with extensive crystalluria indicates acute urinary obstruction leading to renal failure (Maher, 1976).…”

Section: Resultsmentioning

confidence: 99%

Changes in Urine in Experimental Ethylene Glycol Intoxication in Cow Calves

Singh¹,

Kumar²,

Sharma³

1995

Journal of Applied Animal Research

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“…The ethylene glycol molecule itself is not directly responsible for toxicity, which is caused principally by the metabolite glycolate (glycolic acid) [2,4,. This is formed from ethylene glycol via glycoaldehyde by the action of alcohol dehydrogenase (Fig.…”

Section: Discussionmentioning

confidence: 99%