Eustachian Tube Dysfunction after Tobacco Smoke Exposure

Dubin, Marc G.; Pollock, Hoke W.; Ebert, Charles S.; Berg, Eric; Buenting, John E.; Prazma, Jiri

doi:10.1067/mhn.2002.121320

Cited by 30 publications

(16 citation statements)

References 9 publications

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“…The results obtained for all immittance measurements proved consistent with the normality values on guinea pigs, which showed that tobacco smoke exposure alters the Eustachian tube, contributing to the development of otitis media 22,23 corroborate the findings, as well as research in humans which indicated passive smoking as a predominant risk factor for recurrent otitis in children [24][25][26] . No significant differences were found in the comparative analysis of the other variables investigated: equivalent volume of external acoustic meatus, static admittance of peak and gradient, indicating that they do not provide information about the habit of smoking.…”

supporting

confidence: 75%

Estudo comparativo das medidas de imitância acústicas em tabagistas e não tabagistas

Mourão

Baeck

2014

Rev. CEFAC

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OBJETIVO: realizar um estudo comparativo das medidas de imitância acústica em tabagistas e não tabagistas, visando identificar a ocorrência de um comportamento específico na população tabagista.MÉTODOS: em uma amostra 80 indivíduos, 40 tabagistas e 40 não tabagistas, foram obtidos o volume do meato acústico externo, a admitância, a pressão de pico e o gradiente.RESULTADOS: os resultados mostraram que pressão de pico apresentou diferença significante entre os grupos, revelando-se mais negativa no grupo dos tabagistas, tanto para a orelha direita (p=0,004) quanto para a esquerda (p=0,011). Nas demais medidas investigadas não foram verificadas diferenças estatísticas.CONCLUSÃO: Desta forma, o presente estudo conclui que a pressão do pico do timpanograma é uma medida imitanciométrica capaz de diferenciar tabagistas e não tabagistas, sendo que nos tabagistas a pressão mostra-se significantemente mais negativa.

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supporting

confidence: 75%

Estudo comparativo das medidas de imitância acústicas em tabagistas e não tabagistas

Mourão

Baeck

2014

Rev. CEFAC

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“…Long-term passive smoking may result in increased mucous secretion into the eustachian tube, as demonstrated in rats over a 6-month period of exposure; when combined with smoke-induced impaired mucociliary clearance of the eustachian tube, obstruction may occur resulting in middle ear disease and OME [ 5 , 20 , 24 ]. Indeed, further studies in rats have found that tobacco smoke exposure results in prolonged mucociliary clearance times and an inability to equilibrate negative middle ear pressure with increasing smoke exposure [ 27 ].…”

Section: Discussionmentioning

confidence: 99%

Tobacco Smoke Exposure and Eustachian Tube Disorders in US Children and Adolescents

et al. 2016

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ObjectivesTo describe the association between active, environmental tobacco smoke (ETS) exposure and the prevalence of eustachian tube dysfunction (ETD) in the U.S. pediatric population.Study DesignCross-sectional.SettingU.S. representative demographic and audiometric data from the National Health and Nutrition Examination Survey (NHANES);2005–2010.Subjects and MethodsThe study consisted of 2,977 children aged 12–19 years. ETD was defined as middle ear pressure <100mm H20. ETS was defined as non-active smoking in individuals with serum cotinine over the limit of detection (≥0.015 ng/mL) and <10 ng/mL(N = 1559).ResultsThe prevalence of ETD was 6.1%. After multivariate adjustment for age, sex, body mass index, education level, ethnicity, or having a cold, sinus problem or earache during the last 24 hours, compared to unexposed children, the odds ratios (95% confidence interval) of ETD for those exposed to ETS ages 12–15 in the first, second and third tertile of cotinine concentrations were, respectively, 1.38 (0.53–3.60), 0.99 (0.53–3.60) and 2.67 (1.12–6.34). Similarly, the odds ratios (95% confidence interval) of ETD for those exposed to ETS ages 16–19 in the first, second and third tertile of cotinine concentrations were, respectively, 1.28 (0.48–3.41), 0.99 (0.40–2.48) and 2.86 (1.19–6.88).ConclusionThese data suggest that children and adolescents exposed to high concentrations of ETS may have an increased prevalence of ETD.

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“…Passive smoking and environmental pollution can induce inflammation of the mucosal surfaces of the nasopharynx, eustachian tube, and middle ear and might alter immune defenses of the respiratory tract, rendering them more vulnerable to viral and bacterial infection episode [35].…”

Section: Passive Smoking and Environmental Pollutionmentioning

confidence: 99%

Risk factors for the development of otitis media

Dhooge

2003

Curr Allergy Asthma Rep

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Otitis media (OM) is the most frequently diagnosed disease in infants and young children. Large, prospective studies suggest an increase in incidence of this disease during the past 10 to 20 years, possibly reflecting a change in host and environmental risk factors for the development of OM. Good knowledge of host (intrinsic) and environmental (extrinsic) risk factors for the development of otitis media is important in identifying a child at risk for recurrent and persistent OM. This could result in primary and/or secondary prevention of OM and a decrease in complications and sequelae.

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Eustachian Tube Dysfunction after Tobacco Smoke Exposure

Abstract: ET dysfunction after tobacco smoke exposure may predispose children to the development of otitis media.

Cited by 30 publications

References 9 publications

Estudo comparativo das medidas de imitância acústicas em tabagistas e não tabagistas

Estudo comparativo das medidas de imitância acústicas em tabagistas e não tabagistas

Tobacco Smoke Exposure and Eustachian Tube Disorders in US Children and Adolescents

Risk factors for the development of otitis media

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