Focal areas of increased echogenicity within the brain.

Slovis, Thomas L.; Shkolnik, Arnold; Haller, Jack O.

doi:10.7863/jum.1989.8.1.31

Cited by 6 publications

(6 citation statements)

References 4 publications

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“…The causes of hyperechoic cerebral masses seen on a neonatal brain ultrasonograms include intraparen-chymal hemorrhage, infarct, neoplasm, postinfectious calcification, tuberous sclerosis, arteriovenous malformations, and periventricular leukomalacia. 8 In the present case, the early appearance of the parenchymal mass (day 2), the initial homogeneous echogenic appearance of the mass, the subsequent central degeneration after 1 week, and the ventricular involvement are findings characteristic of grade IV intracranial hemorrhage. A later onset and an initial central hypoechogenicity are typical of Candida infection.…”

Section: Discussionsupporting

confidence: 50%

“…8 In the present case, the early appearance of the parenchymal mass (day 2), the initial homogeneous echogenic appearance of the mass, the subsequent central degeneration after 1 week, and the ventricular involvement are findings characteristic of grade IV intracranial hemorrhage. A later onset and an initial central hypoechogenicity are typical of Candida infection.…”

Section: Discussionmentioning

confidence: 82%

See 1 more Smart Citation

Candida brain abscess: a sonographic mimicker of intracranial hemorrhage

Johnson

Kazzi²

1993

Journal of Ultrasound in Medicine

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Section: Discussionsupporting

confidence: 50%

Section: Discussionmentioning

confidence: 82%

Candida brain abscess: a sonographic mimicker of intracranial hemorrhage

Johnson

Kazzi²

1993

Journal of Ultrasound in Medicine

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“…At some 155 J Ultrasound Med 18:153-158, 1999 ASHRAF ET AL point in the course of their evolution, hemorrhage, infarction, and periventricular leukomalacia are seen as hyperechoic areas. 9,10 These abnormal echogenic regions must be differentiated from structures that are normally of greater echogenicity than the surrounding brain tissue. The choroid plexus and meninges, for example, are normally highly echogenic.…”

Section: Discussionmentioning

confidence: 99%

Variation in echogenicity of the basal ganglia: anisotropic effect.

Ashraf

Feldstein

Filly

1999

Journal of Ultrasound in Medicine

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We observed that the fetal brain demonstrates relatively increased echogenicity of the basal ganglia compared with the thalami and cortical brain parenchyma, which we did not observe on neonatal sonograms. We hypothesized that the difference in relative echogenicity was due to differences in imaging techniques and anisotropic effects for prenatal and postnatal brain images. In 18 consecutive neonates, we obtained coronal images of the basal ganglia and thalami through the anterior fontanelle and axial images through the anterolateral fontanelle with both 5 and 7.5 MHz transducers. Two observers determined whether increased echogenicity or conspicuity of the basal ganglia was present, comparing the axial and coronal planes. We observed relatively increased echogenicity of the basal ganglia in the axial plane in 11 of the 16 examinations in this series. Of these 11, the increased echogenicity effect was manifest only in the axial plane in seven neonates. In the four instances in which the increased basal ganglia echogenicity was seen in both the coronal and axial planes, the effect was better shown in axial plane in all four. We did not observe any cases of increased echogenicity of the basal ganglia only in the coronal plane. The increased echogenicity was more conspicuous with the lower frequency transducer in 10 of the 11 examinations. We believe that the change in echogenicity of the basal ganglia is predominantly an anisotropic effect. Observing that increased echogenicity of the basal ganglia can disappear or decrease when comparing images in the axial to the coronal plane or be better demonstrated with lower frequency transducers might be a means by which to distinguish this phenomenon from true pathologic processes of the neonatal brain.

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“…Normal confinement of the plexus echogenicity to the second trimester helps to detect diseases that cause focal areas of increased echogenicity in late fetal life. Slovis et al (1989) reviewed these disease entities, the earliest of which presents at about the 26th week of gestation. Hypoechoic indentations on the plexus, which are interpreted as pseudocysts by some workers, were seen but disregarded in this study, using the method of Nelson et al (1992) to differentiate between a true cyst and a relative sonolucency representing the corpus striatum.…”

Section: Discussionmentioning

confidence: 99%

Echogenicity of the fetal telencephalic choroid plexus as a second trimester developmental landmark: A preliminary report

1995

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Echogenicity of the normal fetal telencephalic choroid plexus was characterized anatomically in 102 consecutive antenatal abdominal ultrasonographic examinations. Echogenicity of the plexus was observed only during the second trimester, first appearing at week 13 and disappearing after week 23. We suggest that this echogenicity may be due to different stages of glycogen chemistry in the plexus. The knowledge that telencephalic echogenicity is confined to the second trimester should help detect diseases that cause focal areas of echogenicity in fetal life.

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Focal areas of increased echogenicity within the brain.

Cited by 6 publications

References 4 publications

Candida brain abscess: a sonographic mimicker of intracranial hemorrhage

Candida brain abscess: a sonographic mimicker of intracranial hemorrhage

Variation in echogenicity of the basal ganglia: anisotropic effect.

Echogenicity of the fetal telencephalic choroid plexus as a second trimester developmental landmark: A preliminary report

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