From Bradycardia to Tachycardia: Complete Heart Block

Kearney, Kathleen; Ellingson, Sonja; Stout, Karen; Patton, Kristen K.

doi:10.1016/j.amjmed.2015.04.013

Cited by 4 publications

(7 citation statements)

References 10 publications

(8 reference statements)

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“…In patients with second degree type II atrioventricular block is common a slowing of impulse conduction on His-Purkinje system, this is demonstrated during electrophysiological study by an abnormally long H-V interval [ 41 ]. High-grade atrio-ventricular block and complete heart block represent the most severe forms of atrio-ventricular nodal disease and are usually associated with underlying heart disease [ 42 ].…”

Section: Pathophysiology Of Bradyarrhytmias In Hfmentioning

confidence: 99%

Management of Arrhythmias in Heart Failure

Masarone

Limongelli

Rubino

et al. 2017

JCDD

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Heart failure patients are predisposed to develop arrhythmias. Supraventricular arrhythmias can exacerbate the heart failure symptoms by decreasing the effective cardiac output and their control require pharmacological, electrical, or catheter-based intervention. In the setting of atrial flutter or atrial fibrillation, anticoagulation becomes paramount to prevent systemic or cerebral embolism. Patients with heart failure are also prone to develop ventricular arrhythmias that can present a challenge to the managing clinician. The management strategy depends on the type of arrhythmia, the underlying structural heart disease, the severity of heart failure, and the range from optimization of heart failure therapy to catheter ablation. Patients with heart failure, irrespective of ejection fraction are at high risk for developing sudden cardiac death, however risk stratification is a clinical challenge and requires a multiparametric evaluation for identification of patients who should undergo implantation of a cardioverter defibrillator. Finally, patients with heart failure can also develop symptomatic bradycardia, caused by sinus node dysfunction or atrio-ventricular block. The treatment of bradycardia in these patients with pacing is usually straightforward but needs some specific issue.

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Section: Pathophysiology Of Bradyarrhytmias In Hfmentioning

confidence: 99%

Management of Arrhythmias in Heart Failure

Masarone

Limongelli

Rubino

et al. 2017

JCDD

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“…Complete heart block is characterized by complete dissociation of atrial and ventricular electrical conduction, such that no electrical signals generated above the AV node (particularly the sinus node) are conducted to the ventricles. Typical junctional or ventricular escape rhythms result in pulse rates from 30 to 50 beats per minute 7 …”

Section: Intrinsic Vs Extrinsic Causesmentioning

confidence: 99%

“…Gascho and Schieken 10 and Nikolic et al 11 reported ventricular tachycardia, including torsades de pointes, and ventricular fibrillation in 2 children with congenital CHB. Each child had a prolonged QT interval, which is a complication of bradycardia, and increases the likelihood of sudden death, in addition to the CHB 7,12 . Nikolic et al 11 observed that CHB confers a greater tendency for arrhythmias with advancing age.…”

Section: Intrinsic Vs Extrinsic Causesmentioning

confidence: 99%

“…Typical junctional or ventricular escape rhythms result in pulse rates from 30 to 50 beats per minute. 7 Complete heart block can be congenital or acquired. Congenital CHB is defined as AV dissociation and a slow ventricular rate coexisting early in life without an infective or surgical cause.…”

Section: Intrinsic Vs Extrinsic Causesmentioning

confidence: 99%

“…Each child had a prolonged QT interval, which is a complication of bradycardia, and increases the likelihood of sudden death, in addition to the CHB. 7,12 Nikolic et al 11 observed that CHB confers a greater tendency for arrhythmias with advancing age.…”

Section: Intrinsic Vs Extrinsic Causesmentioning

confidence: 99%

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Lesions of the Cardiac Conduction System and Sudden Death

Cohle,

Wygant

2023

Am J Forensic Med Pathol

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When a young previously healthy person dies suddenly, occasionally, the scene is noncontributory and the autopsy and drug screen are negative. In such cases, additional studies, including genetic assessment and cardiac conduction system examination, should be performed. We performed a literature search and reviewed our own material to identify possible or definite conduction system anomalies that may cause death. We identified intrinsic conduction system disease including cystic tumor of the atrioventricular node, atrioventricular node (cystic tumor of the AV node), and fibromuscular dysplasia of the atrioventricular node artery to be likely causes of death. Extrinsic causes, in which a generalized disease affects the conduction system, include tumors, autoimmune disease, infiltrative disorders, and others, are a second category of diseases that can affect the conduction system and cause atrioventricular block and sudden death.

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