Genesis of the austin flint murmur: Relation to mitral inflow aortic regurgitant flow dynamics

Abstract

The increased velocity of early diastolic mitral inflow in patients with the Austin Flint murmur is due to aortic regurgitation, but rapid mitral inflow is not an essential requirement for production of the murmur. In some cases, the Austin Flint murmur may be generated by aortic regurgitant flow alone.

Altered Mitral Inflow Orifice in Severe Aortic Regurgitation: Real Time Three‐Dimensional Echocardiographic Findings

“…While the cause of the Austin Flint murmur has been subject to debate (with one study finding no correlation between the presence of this murmur and mitral valve area), this case seems to endorse the hypothesis that early diastolic closure of the AML induced by the aortic regurgitant jet, even without hemodynamic significance, is pivotal in causing this particular auscultatory finding.…”

Unraveling a rare presentation of bivalvular infective endocarditis using POCUS

“…The regurgitant jet can be directed towards the mitral valve, leading to the development of vegetations or perforation of the AMVL, resulting in significant mitral regurgitation and bivalvular endocarditis. Sometimes, the same aortic regurgitation “backward‐jet” hitting the AMVL causes AMVL fluttering and turbulence in the left ventricle leading to a rumbling diastolic murmur called “Austin‐Flint murmur” that is best heard at the apex 6 . Another important cause of bivalvular endocarditis may be the spread of infection into the adjacent mitral annulus, resulting in secondary destruction of the mitral annulus.…”

Concealed Left Ventricular Hypertrophy and Diastolic Dysfunction in Hypertrophic Cardiomyopathy in the Presence of Acute Left Ventricular Volume Overload. A Case Report.

“…Acute severe aortic regurgitation has hemodynamic characteristics distinctly differ from those of chronic aortic regurgitation. [3][4][5] In acute regurgitation, massive regurgitation rapidly flows into the left ventricular cavity in early diastole. As a result, left ventricular pressure becomes equal to aortic pressure, and exceeds the left atrial or pulmonary capillary wedge pressure by mid-to , late diastole, leading to early termination of the diastolic transmitral inflow.…”